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Pharmacology-PA Test

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1.
77. What is the standard dosage and high risk pt dosage?
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68. What are the ADRs of heparin?
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138. Compare ticlopidine with aspirin?
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37. What is mild hemophilia A?
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98. Is it well absorbed orally?
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56. What is MOA for heparin?
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45. How is life threatening bleeds treated?
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127. What is abciximab indicated for?
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9. Are folic acids sensitive to meals?
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55. Where is heparin obtained from?
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12. Where is B12 absorbed?
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33. What else does vWF bind to?
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107. What should be considered when treating Lepirudin?
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112. What results in binding of fibrinogen?
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80. What are the considerations when using protamine sulfate?
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115. What are some examples of platelet aggregation inhibitors?
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25. What is the second degree hemostasis?
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70. How is aPTT measured?
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142. What is MOA for Dipyridamole?
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73. What are the indications for heparin?
A.
Inhibits thromboxane A2, synthesis and may decrease platelet adhesion through potentiation of prostacyclin PG12
B.
Heparin catalyzes inactivation of thrombin and factor Xa prolonging aPTT
C.
Yes, after a meal a person may appear to have enough folic acid, may not seem like Folic acid anemic
D.
Exposed collagen binds to vWF
E.
Adjust with renal compromise
F.
Clotting plasma, fibrin deposition
G.
Bleeding episodes, thrombocytopenia, pruritis, hypersensitivity rxns, chills, fever, urticaria, anaphylaxis
H.
Blood is collected with oxalate or citrate to prevent coagulation, specimen is delivered to the lab. Activator (such as silica, celite, kaolin, ellagic acid) is added to activate intrinsic pathway and calcium is added to reverse the effect of oxalate. The
I.
Ticlopidine is more effective than aspirin in preventing initial stroke in pts with stoke precursors, but causes significantly more ADR than asprin.
J.
Prevent extension of DVT, pulmonary embolism, coronary thrombosis, in pt with artificial heart valves or suffering from stroke
K.
Yes, but takes 4-5 days for full effect
L.
Problems with bleeding only after serious injury or trauma or surgery
M.
Linkage of activated platelets through fibrinogen leads to aggregation
N.
Absorbed in the ileum
O.
5000units
P.
Reserved for major bleeding due to risk of anaphylaxis
Q.
As an adjunct to aspirin and heparin for the prevention of acute cardiac ischemic complications not responding to conventional medical therapy or when percutaneous coronary intervention is planned w/in 24hrs
R.
100unites/kg porcrine F8 followed by IV infusion of 4 units/kg/hr
S.
Aspirin, ticlopidine, dipyridamole, clopidogrel, sulfinpyrazone
T.
porcine or bovine intestinal mucosa.
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21.
3. How long does it take before Hgb rise is noticed after Fe treatment?
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22.
89. What is the protamine sulfate effect on LMWH?
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76. When is heparin clearance prolonged?
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24.
4. What happens if you take Fe with meals?
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25.
72. What is administered in heparin over dose?
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26.
15. What produces EPO?
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27.
108. Platelet activation and degranulation result in local accumulation of what molecules?
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28.
31. What happenes to factor 8 when not bound vWF?
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29.
Functional measure of intrinsic pathway
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30.
A modified version of EPO which has an increased half life

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