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HCC 2008 Diabetic Complications

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Answer
Diabetic complications can   result in permanent disability and/or death  
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Acute diabetic complications   hypoglycemia, diabetic ketoacidosis(DKA), hyperglycemic hyperosmolar non ketotic syndrome(HHNS), hypoglycemia Insulin reaction, blood glucose<50-60 mg/dl  
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mild symptoms   sweating, tremors, tachycardia, palpation, nervousness, hunger  
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moderate symptoms   inability to concentrate, lightheadedness, confusion, irrational or combative behavior, double vision, drowsiness  
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severe symptoms   disoriented behavior, seizures, difficulty arousing from sleep  
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Treatment of hypoglycemia   aimed at prevention, STAT bloog glucose, 15g of quick acting CHO equivalent to 2-3 tsp of sugar or honey, 6-10 lifesavers, 4-6 o of fruit juice, if unconscious Glucagon 1mg SC or IM of 25-50 ml 50% dextrose in water(D50W)  
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Patient teaching Hypoglycemia   meal pattern, insulin administration, exercise, snacks, routine SBGM, wear ID bracelet, s/s of hypoglycemia instruct client on s/s and treatment, instruct on balancing of exercise and adjusting insulin  
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Concerns in elderly diabetics   skipping meals, decreased visual activity(with too much insulin), living alone, decreased renal function ***should slow functions: dont rid of insulin too fast  
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Diabetic Ketoacidosis(DKA) is also know as a   diabetic coma  
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cause(s) diabetic ketoacidosis   missed dose or too little insulin, illness or infection, undiagnosed and intreated DM  
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symptoms of ketoacidosis   dry skin and mucous membrane, high blood sugar, ketoacidosis-mainly type 1  
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Three key factors of DKA   hyperglycemia, dehydration and electrolyte loss, metabolic acidosis  
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what happens during DKA   bodies cells cannot obtain sufficient fuel glucose, they burn protein and fat for the energy that need which leeds to DKA, the fat burning leads to the formation of highly acidic KETONES  
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difference btw DKA type 1 and type 2   type 2 diabetics(NIDDM) have enough insulin to prevent the breakdown of fats ad don't produce KETONES  
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Clinical manifestations DKA   blurred vision, WEAKNESS, HEADACHE, dry skin, dry mucous membranes, orthostatic hypotension, dehydrations, anorexia, nausea, vomiting, deep labored(kussmaui resp.), acetone breath, comatose, confused, irritable, tachycardia  
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Assessment and Diagnostic findings of DKA   glucose level>300 (can be>1200)mg/dL, serum bicarbonate low, low pH, PCO2 high or no change, Na & K+ normal or high, high creatinine, BUN, High Hct bc of dehydrated  
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if you are sick particularly vomiting have diarrhea or a fever and diabetic you blood sugar will ___________   rise; you must call the doctor and still take your insulin  
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when you are sick you must ________ stop your insulin, in fact you might have to give _______insulin   not; more  
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if sick ___________ self monitoring and test ever ___________hrs   increase; 3-4  
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test your urine for _________ Q____hrs   acetone; 3-4  
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when sick try to eat ___________ meals, if vomiting etc you need to drink _______ liters of fluid (water tea juice salty soup)   normal; 2-3  
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remeber if sick report N/V to _______ _________ ____________   health care provider  
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renal failure causes __________ of ketones and glucose and result is continued acidotic state   retention  
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DKA treatment-Goal   immediate intervention (as nurse start IVs), manage dehydration, restore electrolytes, reverse acidosis  
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Monitoring dehydration with fluid replacement includes   administer .9 Nacl or 0.45% Nacl at a rapid rate, glucose level 250 give D5w, monitor I/O, observe for orthostatic changes in B/P and P, may have to give plasma expanders, observe for fluid overload  
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restoring electrolytes during DKA includes   monitoring e-lytes especially K+, administer insulin( low dose IV), administer potassium  
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Reversing acidosis DKA includes   measuring blood glucose hourly, adminiter insulin at slow rate 5U/hr, hypertonic solutions  
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a patient with DKA would receive solution at what blood glucose level?   250  
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why would a DKA pt need more then one IV line?   to flush fluids in one arm and insulin in another, can give fluid at a higher rate then insulin  
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nursing management of DKA   monitor: electrolytes, blood glucose, I/O administer: fluids, insulin, and other meds, prevent fluid overload, assess renal function before administering K+  
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HHNS, HHS occur when   occurs when there is not enough insulin to prevent hyperglycemia, osmotic diuresis and extracellular fluid depletion but enough insulin to prevent DKA  
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HHNS, HHS occurs most often in   patients with indiagnosied diabetes or patient with mild type II diabetes, may be precipitated by stress or other illness, older adults, certain medications  
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Symptoms of HHNS   profound dehydration, hypotension, tachycardia and neurologic signs such as... high urine volume with causes dehydration and hyperosmolar blood + fruity odor in mouth  
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Assessment and Diagnostic findings in HHNS   BG 600-1200 mg/dL, serum osmolarity> 350mOsm/L, mental status changes, focal neurological deficits- aphasia hemiparesis, ataxia, hallucinations, postural hypotension, dehydration  
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Differences btw DKA and HHNS   DKA more common in type 1 HHNS more common in type 2, DKA has a rapid onset and HHNS is slower, glucose level for DKA is >250 HHNS is typically >600, DKA pH 7.3 HHNS pH is normal, ketones are present in DKA not in HHNS  
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medical management of HHNS   rapid IV infusion of 0.9 NS or 0.45 NS(initially) then dextrose, monitor and replace electrolytes, regular insulin IV  
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Nursing Implications of HHNS   cardiopulmonary monitoring, I&Os, frequent VS, fluid status, laboratory values, maintain safety/prevent injury, treat underlying precipitating condition  
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In summary collaborative care of DKA includes   diagnostic tests, blood glucose, CBC, ketones(blood and urine), pH, e-lytes, BUN, serum osmolality, IV insulin, IV fluids> HHNS, e-lye replacement, mental status, glucose monitoring, I/O, blood and urine, ketones, EKG monitoring, CV and resp monitoring  
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In summary collaborative care of HHNS includes   diagnostic tests, blood glucose, CBC, ketone, pH-normal, BUN, serum osmolality >350, IV insulin IV fluids, e-lytes replacement, mental status, glucose monitoring, I/O, blood & urine, ketone(usually no ketoacidosis), EKG monitoring, CV & Resp monitoring  
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nursing planning and implementation or macrovascular complications includes   prevention and treatment of risk factors of atherosclerosis, diet and excercise to manage obesity, hypertension and dyslipidemia, smoking cessation, control blood glucose, knowing s/s, cut alcohol, regular eye and dental exams, hemoglobin A1C  
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Microvascular Long term complications includes   diabetic retinopathy, microvascular damage to retina due to chronic hyperglycemia, almost always present in pts with type 1 diabetes btw the ages of 10-15 yrs, leading cause of blindness in ages 24-74  
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other ocular complications include   cataracts, lens changes, extrocular muscle palsy, glaucoma  
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clinical manifestations of microvascular complications and retinopathy   blurred vision- red or black spots, muscular edema, hemorrhage, complete loss of vision  
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Assessment and Diagnostic findings of microvascular complications and retinopathy   flouicein angiography- use of dye to examine retinal vessels; manage with laser photocoagulation or vitrectomy  
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nursing planning and implementation of microvascular complications   tight control of blood glucose, control of ypertension with ACE inhibitors, prevent and.or treat UTIs early, avoid nephrotoxic drugs, low sodium, low protein diets, smoking cessation, regular eye exams and renal screenings, refer to low-vision centers  
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Neuropathy   decreased pain and pressure sensation, increased dryness and fissuring leas to risk for injury and infection  
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Peripheral vascular disease   poor circulation contributes to poor wound healing and gangrene---loss on sensation  
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Immunocompromised   hyperglycemia impaire the ability to leukocytes to destroy bacteria. Therefore there is a lowered resistance to infection  
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Diabetic foot care---how to prevent complications?   inspect feet when you take off socks, look for small injuries and redness, wash feet everyday in luke warm water, dont soak no water higher the 37C, keep feet clean and dry, check shoes b4 putting them on, never walk barefoot  
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Diabetic foot care---more ways to prevent complications?   not hot water bottles or devices to warm feet, if you have an injury call doctor, if skin is dry use neutral creams, if humid use powder, file nails instead of cutting  
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Management of Diabetes---goals   to normalize activity and blood glucose levels, reduce the development of complications, maintain as normal a lifestyle as possible  
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