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Addison's Disease and Cushing's Syndrome

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Question
Answer
Cushing Syndrome   excess of corticosteroids  
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causes of Cushing's Syndrome   iatrogenic administration of exogenous corticosteroids ACTH secreting pituitary adenoma adrenal tumors ectopic ACTH production by tumors (lungs / pancreas  
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clinical manifestations with excess gluco-corticoids   hyperglycemia, hypokalemia, hypocalcemia, HTN, hypervolemia, depression, emotional irritability, loss of collagen, muscle waste, weight gain, thinning of hair, red cheeks, buffalo hump, moon face and slow wound healing  
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mineralcorticoid excess CMs   hypokalemia, HTN  
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androgen excess CMs   severe acne, virilization in women, feminization in men  
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diagnostic studies for Cushing's Syndrome   midnight or late night salivary cortisol low dose dexamethasone suppression test 24 hr urine cortisol: > 100 mcg  
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if cause is iatrogenic- medical management   gradually discontinue therapy, decrease dose, convert to alternate day regimen  
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nursing management of Cushing's Syndrome   monitor: vitals, daily weight, glucose, electrolytes emotional support  
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what to watch out for with electrolytes   calcium (weak), increases risk of fractures and falls  
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pre-op for surgical removal   correct hypertension, hypokalemia, hyperkalemia, hyperglycemia  
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post op care for surgical removal   risk for hemorrhage, monitor BP, fluid balance, electrolytes, HR, I&O, RR, weight, vomiting, administer SoluCortef  
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education for Cushing's   wear medic alert bracelet, avoid exposure to extreme temperatures, infection and stress, how to adjust meds in relation to stress when to call HCP- SOB, dizziness, vomiting  
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primary addison's disease   lack of glucocorticoids, mineralcorticoids, and androgens  
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secondary addison's disease   lack of pituitary ACTH, lack of glucocortoids and androgens  
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autoimmune polyglandular syndrome   most common in white females co-occurring endocrine conditions: type 1 diabetes, autoimmune thyroid disease, pernicious anemia, celiac disease  
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causes of addison's disease   TB, amylodosis, fungal infections, AIDS, metastatic cancer adrenal hemorrhage, chemo, ketoconazole therapy for AIDS, bilateral adrenalectomy  
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clinical manifestations of Addison's Disease   insidious onset- anorexia, nausea, progressive weakness, fatigue, weight loss, hyperpigmentation, depression/irritability, vitiligo, hypoglycemia, hyperkalemia, hyponatremia (craving salt), hypovolemia (dizziness, headache), hypercalcemia (joint pain)  
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addisonian crisis   actual adrenal insufficiency, sudden sharp decrease in hormones, life-threatening  
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triggers of addisonian crisis   stress, stopping medications, adrenal surgery, sudden pituitary gland destruction  
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clinical manifestations of addisonian crisis   sudden pain (in back / legs), syncope, shock. hypotension, severe vomiting and diarrhea, headache  
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ACTH stimulation test   baseline levels of cortisol and ACTH IV injection of synthetic ACTH levels rechecked 30 and 60 mins if little or no increase of blood cortisol levels then addison's is suspected  
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CRH stimulation test   abnormal ACTH test response IV injection of synthetic CRH blood drawn 30 and 60 mins after High ACTH with no cortisol --> addison's  
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Diagnostics for Addison's   high K+, decreased chloride, sodium and glucose EKG changes, CT scan / MRI  
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medical management of Addison's   hydrocortisone (both mineral and glucocorticoid) (increase during stress) Fludrocortisone (Florinef) increase dietary salt intake  
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addisonian crisis management   shock management, high dose hydrocortisone replacement, 0.9% saline solution and 5% dextrose  
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nursing management for Addison's   correct fluid and electrolyte balance - vitals, ECG monitoring, neurologic status, daily weight, I&O monitor blood glucose monitor nutritional status (increase protein, carbs, fiber and fluids) protect from extremes (light, noise, temperature)  
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patient teaching for addison's   corticosteroid dosing corticosteroid deficiency / excess signs and symptoms wear med-alert bracelet increase dose during times of stress  
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excess corticosteroid symptoms   vomiting and diarrhea  
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glucocorticoid dosing   2/3 in AM, 1/3 in afternoon  
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mineralcorticoid dosing   1x daily in AM teach how to take BP at home increase salt intake  
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corticosteroid side effects   decreased potassium, calcium increased glucose and BP delayed healing suppressed immune response peptic ulcer disease muscle atrophy and weakness protein depletion risk for adrenal crisis if stopped abruptly  
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expected effects of corticosteroid therapy   suppression of inflammation immunosuppression maintenance of BP  
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dietary needs for addison's   increase protein, 1500 mg Ca, low in fat, no simple carbs  
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when to notify HCP   if blood glucose is > 120, if epigastric pain occurs without relief from antacids  
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other patient teaching with corticosteroid therapy   sodium restriction when edema occurs, rest/ exercise - daily nap recommended prevent injury and infection take in morning with food do not stop abruptly monitor for hyperglycemia  
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