NEURO ....C46 Test
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| A. OD, INFX, HYPOTHYROID, DEHYDRATION, HEART DISEASE, STROKE, COPD, B. to decrease inflammation & suppress the immune systemC. UNKNOWN, BUT FAMIAL CASES HAVE BEEN REPORTEDD. DOCUMENTED PRESENCE OF DEMENTIA W/ONSET B/W 40 & 90 YRS OLD, NO LOSS OF CONSCIOUSNESS & ABSCENCE OF SYSTEMIC OR BRAIN DISORDER THAT COULD CAUSE MENTAL CHGS E. UNKNOWN = AMYLOID PLAQUES & NEUROFIBRILLARY TANGLES ARE SEEN IN AUTOPSYF. relaspsing - remitting; primary progressive; secondary progressive; & progressive relapsing G. MRI w/findings of lesions is most definitive test; also CSF analysis for elevated IGg, ct scan = enlrgd ventricles , atrophy & white matter lesions; areas of chgs in glucose metabolism from PET scan, H. WANDERIING, PERIODS OF LUCIDITY, SUNDOWNING, LANGUAGE DEFICITS, APRAXIA I. APRAXIA,ASTEREOGNOSIS (INABILITY TO IDENTIFY OBJECTS BY TOUCH), AGRAPHIAJ. VASCULAR DEMENTIA , THE 2ND MOST COMMON CAUSE OF DEMENTIAK. ABNORMAL TAU PROTEINS BUILT UP IN NEUROFIBRILLARY TANGLES = FRONTAL & TEMPORAL LOBESL. COGNEX(TACRINE), ARICEPT (DONEPEZIL), EXELON (RIVASTIGMINE),(PARASYMPATHOMIMETICS)M. INCONTINENCE, FALLS, DELUSIONS, PARANOIA, ANOREXIA, N. thioridazine (melaril), or haloperidol (haldol) O. RULE OUT ANY OTHER POSSIBILITIESP. visual, auditory or somatosensoryQ. APPEARS HEALTHY & ALERT; RESTLESS, FORGETFUL OR UNCOORDINATED; DISORIENTED TO TIME & DATE; PROBLEMS W/SIMPLE CALCULATIONS |
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