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nsg 210 ch 24 respir
NSG 210 ch 24 respiratory
Question | Answer |
---|---|
What is COPD? | Disease characterized by airflow limitation that is not fully reversible and abnormal inflammatory response to noxious particles or gases. |
what 2 diseases make up COPD? | Emphysema and Chronic bronchitis. |
When do people with COPD usually become symptomatic? | middle adult years. |
In COPD where does the inflammatory response occur? | Proximal Airways ( trachea and bronchi) Peripheral airways ( bronchioles), lung parenchyma and pulmonary vasculature. |
What is bronchitis? | Disease in which there is the presence of cough and sputum for at least 3 months in each of 2 consecutive years. |
What causes bronchitis? | Irritants cause the bronchials to inflame and narrow and the same irritants also cause mucous secreting cells and goblet cells to produce more mucous further narrowing the airway. |
When are acute exacerbations of chronic bronchitis most likely to happen? | During the winter. |
What are the clinical symtoms of bronchitis? | Productive cough, decreased exercise tolerance, prolonged expiration adn cyanosis with peripheral edema (blue bloater). |
Medical mngmt of bronchitis? | Stop smoking, decrease irritants,antibiotics, increase fluids, corticosteriods, bronchodialators. |
What are the clinical symtoms of emphysema? | Accessory muscle use, barrel chest, pink color (pink puffer). |
What part of the lung does emphysema effect and how? | It effects the alveoli by over distention prohibiting gas exchange. |
Complications of emphysema? | Increased CO2 leading to repiratory acidosis and right sided heart failure. |
What are the risk factors for COPD? | Exposure to tobacco smoke, Passive smoking, occupational exposure ( dust and chemicals), ambient air pollution, genetic abnormalities (antitrypsin deficiency). |
What is spirometry used for? | Evaluate airflow obstruction determined by the ratio between FEV to FVC ( forced vital capacity). |
What are the complications of COPD? | Respiratory insufficiency and failure, pneomonia, chronic atelectasis, pneomothorax and pulmonary arterial hypertension. |
What would you include in your health history and assessment of a pt. with COPD? | Exposure to risk factors, Hx of COPD or respiratory problems, length of time for repiratory difficulty, is there exercise, smoking HX, exposure to smoke. |
What is the single most cost effective intervention to reduce the risk of developing COPD? | STOP SMOKING!!!! |
What is the phamacologic therapy for COPD? | Bronchodialaters Which relax smooth muscle dialating the airway and Inhaled corticosteriods which decrease inflamation and swelling, diuretics for bronchitis, O2, flu vaccine Q year and pneumovax Q 5 years. |
When giving O2 to a COPD pt. what do you need to watch for? | Giving to much can cause them to stop breathing from the build up of CO2 witch shuts down the respiratory drive. |
Surgical management of COPD? | Bullectomy, Lung volume reduction surgery and lung transplant. |
What is a bullectomy? | Bullae are enlarged airspace that do not contribute to ventilation and impair gas exchange and are surgically removed. |
what is lung volume reduction surgery? | Removal of a portion of the diseased lung parenchyma which allows functional tissue to expand. |
What breathing exercises should you teach a pt. with COPD? | Diaphragmatic breathing which reduces respiratory rate and increases alveolar ventilation. |
What nursing Management would you do for a pt. with COPD? | Monitor for dyspnea and hypoxemia, increasing fluids, use if incentive spirometer, effective coughing, cough turn and deep breathe, pursed lip breathing, pace activities, ambulate pt., monitor LOC and O2 saturation. |
What is brochiectasis? | A chronic irreversable dilation of the bronchi and bronchioles. |
WHAT CAN CAUSE BRONCHIECTASIS? | Airway obstruction, duffuse airway injury, pulmonary infection, cystic fibrosis and idiopathic causes. |
clinical manifestations of bronchiectasis? | Chronic cough, purulent sputum in copious amounts, clubbing of fingers and hemoptysis. |
Medical management of bronchiectasis/ | Promote bronchial drainage by gravity, percussion to loosen secretions and antibiotics if C&S confirms. |
What is Asthma? | Chronic inflammatory disease of the airways that cause hyperresponsivness, mucosal edema and mucus production. |
What is the strongest predisposing factor for asthma? | Allergies |
What types of irritants can set off an asthma attack? | Pollen, mold, dust, cold, heat, perfumes, smoke, exercise. |
What role do mast cells play in asthma? | They release histamine, bradykinin, prostiglandins and leukotrienes which perpetuate inflamation and vasocostriction. |
Clinical manifestations of asthma? | Cough with or without sputum, dyspnea, wheezing, chest tightness, diaphoresis, tachycardia, and widened pulse pressure. |
What is the clinical significance of a normal PaCO2 during an asthma attack? | May signal impending respiratory failure. |
What is the best prevention for asthma? | Undergo test to indentify the substances that precipitate the symptoms. |
Complications of asthma? | Status asthmaticus, respiratory failure, pneumonia, atelectasis and airway obstruction. |
Why would you administer fluids to a person with asthma? | Thin secretions and because people with asthma are frequently dehydrated from diaphoresis. |
Asthma quick relief medications? | Beta 2 agonists such as albuterol, levalbuterol and pirbuterol. |
How do beta 2 agonists work? | They relax smooth muscle causing bronchial dilation. |
Long term asthma control medications? | Inhaled corticosteroids such as beclomthasone, fluticasone. ( look for ending in ide or one). Long term Beta 2 agonists such as salmeterol and formoterol ( look for erol). |
When a pt. is on a inhaled corticosteroid what should you instruct them to do after taking it? | Wash mouth out with water cause they can get thrush. |
Can long term asthma control medications be used for an acute asthma attack? | NO! you must use a short term beta 2 agonist. |
Side effects of corticosteroids? | Cough, thrush, glucose abnormalities, osteoporosis, growth suppression, dermal thinning and cushings syndrome. |
Side effects of short term beta agonists? | Tachycardia, muscle tremor, hypokalemia and headache. |
What would be used in a typical asthma medication regiment? | Short term beta2 agonist for acute episodes, inhaled corticosteroids for inflammation, long term beta2 agonists and leukotriene modifiers for decreased mucus production. |
What interventions would the nurse preform for an asthma pt.? | Obtain Hx of allergic reactions to medications, identify medications pt. is already taking, administer medications and monitor pt. responce and administer fluids if pt. is dehydrated. |
What is peak flow monitoring and why is it used? | A meter that measures the highest airflow during a forced expiration. It is used to measure asthma severity and when added to symptom monitoring indicates the current degree of asthma control. |
What is status asthmaticus? | A severe and persistant asthma attack that does not respond to conventional therapy. |
Clinical manifestations of status asthmaticus? | Labored breathing, prolonged exhalation, wheezing. ( as the obstruction worsens the wheezes may disappear). |
Medical management of status asthmaticus? | Short term beta2 agonist, systemic corticosteroids, O2. |
Nursing management of status asthmaticus? | Actively access airway, fluid intake, monitor B/P and HR. |
What is cystic fibrosis? | Multisystem genetic disease characterized by thick viscous secretions in the lungs, pancreas, liver, intestine and reproductive tract. |
Clinical manifestations of CF/ | Productive cough, wheezing, hyperinflation of the lung fields on chest X ray, chronic respiratory inflammation, sinusitis and nasal polyps. |
Medical Management of CF? | Antibiotics based on results of C&S, airway clearance, effective coughing and supplements of vitamins A,D,E and K. |
Nursing Management of CF? | Help pt. manage pulmonary symptoms, chest physiotherapy, breathing exercises, adequate fluid and dietary intake and to keep away from crowded places to lessen the chance of getting a respiratory infection. |