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TEST #5 F&E
Patho
Question | Answer |
---|---|
what is the purpose of fluids in the body? | transportation, electrical conductions, energy production, nad maintaining homeostatis |
where are fluids in the body found? | intra cellular fluid, extracellular fluid, intravascular and interstitial |
pressure of H2O against the membrane | osmotic pressure |
the pressure produced by or associated with osmosis and dependent on molar concentration and absolute temperature | osmotic pressure |
amoutn of the concentration of the solution | osmolarity, osmolality |
directional movement of the fluid | tonicity |
no movement of fluid and replaces lost fluid | isotonic |
shifts fluid out of the vessles into cells or tissue | hypotonic |
shifts fluid from the cells and tissue into the vessels | hypertonic |
what does a high osmolarity/osmolality mean? | more concentration with less fluid |
What does a low osmolarity/osmolality mean? | a lower concentration with more fluid |
describe the fluid movement of the infusion of isotonic solution into veins | no fluid movement |
describe the movement of the infusion of hypertonic solution into the veins | fluid movement into veins |
describe the fluid movement of the infusion of hypotonic solution into the veins | fluid movement out of the veins |
when fluid is not in vascular space or in the cells. this causes ____ | third space...edema |
> pressure of the capillary in which fluid begins to leak out | capillary filtration pressure |
looking from the vascular side and the plasma proteins keep fluids from shifting into weird places | capillary colloidal pressure |
pressure outside of the vessel, pushing back into the capillary system | interstitial hydrostatic pressure |
looking from the cell side and plasma protins keep fludis from shifting into wierd places | tissue colloidal osmotic pressure |
where does the biggest water fluid intake come from? | water beverages |
water chases ____ and ___ | glucose and Na |
what is the normal concentration of Na? | .9% |
if fluid was hypotonic, what % of saline would be given to a pt? | 0.45% or 1/2 of Normal Saline |
If fluid levels where hypertonic, what % of saline would you give to a pt? | 3% normal saline ex: edema and burned pt |
what part of the loss of water cannot be calculated? | insensable water loss (ex: sweating, lung water from talking and coughing) |
what is the best way to tell if there is a balance or inbalance of fluids? | weigh the pt each day at the same time, with the same clothes on, and the same scale |
what causes the biggest loss of water from the body? | urination |
accumulation of interstitial fluids and can be localized or generalized | edema |
termed used for water edema | hydro edema |
term used for water shift to the belly | ascites edema |
term for fluid in the spaces around membrane | effusion edema |
water makes up ____ of body fluids | 90-93% |
Na makes up ____ of extracellular fluid | 90-95% |
___ regulates the extracellular fluid | Na |
what are the sources of Na? | canned or boxed food, stuff outside cell, and in GI secretions |
how is Na secreted? | through urine and GI tract |
what is the role the kidneys play with Na? | kidneys regulate the amount of Na in the blood stream |
___ holds in Na. Na holds in ____ | aldosterone...water |
there is a high concentration of ___ outside of the cell | Na |
___ triggers dehydration and < volume of water | thrist |
hypodipsia | < thirst |
polydipsia | excessive thirst (ex: in DM pt) |
hormone that prevents you from urination | antidiuretic hormone |
what is diabetes insipidus? | when a pt has to go go go (> urination like in a DM pt) |
what does a SIADH (syndrome of inappropriate antidiuretic hormone) do? | this is when you hold in urination too much |
what are the 3 main causes of hypovoliema (fluid volume deficit)? | insufficient intake and inadequate replacement, and excessive fluid loss |
abnormal deficiency of protein in the blood | hypoproteinemia |
fluid volume deficit | hypovoliema |
fluid volume excess | hypervoliema |
what are the causes of hypervoliema or fluid volume excess? | escessive intake, excessive use of saline edemas, steroid therapy, heart failure, liver failure, stress, remobilization after a burn tx, hypertonic or hyperosmolar solutions |
what are some major s/s of hypervoliema (fluid volume excess)? | tachycardia, hypertension, wt gain, JVD, tachypnea, dyspnea, crackles, cough, peripheral edema, < HCT, < BUN, < specific gravity |
measures osmolarity | specific gravity |
low blood Na | hyponatremia |
having too much Na or water in the blood vessels, which causes an > in the ICF pressure | hypertonicity |
too little Na or water in the blood vessles, causing a < in the pressure of the ICF | hypotonicity |
euvolemic | normal fluid volume |
what are the ways to lose Na out of the GI tract? | GI suction, vomiting, diarrhea |
passage ways the body makes that you were not born with | fistulas |
what are the early s/s of hyponatremia? | n/v/d, abdominal cramps |
what are the main s/s of hyponatremia? | weakness, fatigue, anorxia |
what are the late s/s of hyponatremia? | tremors, seizures, lethargy, mental confusion, disorentation |
high blood Na | hypernatremia |
an excessive loss of water will cause hypernatremia b/c ___ | Na levels will get HIGH if there isn't enough fluid |
what are the major s/s of hypernatremia? | tachycardia, weak pulse, postural HTN, thirst, low grade temp, tachypnea, oliguria, > BUN, > osmolarlity, > Na, > HCT, > RBC |
reduced secretion of urine | oliguria |
is vital to the acid-base balance | Na |
the ___ balance is found inside of the cells | K |
second most abundant cation | K |
major cation of the ICF | K |
___ outside of cell, ____ inside of cell | Na....K |
critical in osmotic and acid-base balance, kidnesy's ability to concentrate urine, necessary for growth, carb, gluscose, and protein metabolism, and electrical conduction | K |
what is the usual source of K intake? | diet |
what are the main sources of excretion of K? | kidneys, stool, sweat |
describe Na and K relationship | inversely proportional= Na up, K down....Na down, K up |
____ shifts between ICF and ECF in attempts to maintain balance | K |
what influences K shift between ICF and ECF | insulin, B-adrenergic stimulation, serum osmolarity, acid-base balance, and exercise |
low K in blood | hypokalemia |
what are the main causes of hypokalemia? | < intake, diuretics |
what are the main s/s of hypokalemia? | orthostatic HTN, muscle weakness and cramping, parasthesia,hyperglycemia, metabolic alkalosis, dyspnea, polyuria, polydipia, ECG changes, and cardiac arrest |
what are the ECG changes in hypokalemia? | flat t wave, presence of U wave, depressed ST segment. prolonged QT and PR interval, dysrhythmiass |
what are the most dangerous s/s of hypokalemia? | cardiac dysrythmias b/c they are a deadly sign |
how should you adm an K IV? | slowly and diluted...not as a push |
high K in blood | hyperkalemia |
what are the main causes of hyperkalemia? | renal impairment, K sparing diuretics, ACE inhibitors, burns |
what are the ECG changes in hyperkalemia? | tall, narrow, peaked T wave...wide QRS...prolonged PR interval...flattened to absent P wave...dysrhythmias (life threatening) |
most of __ is found in the bones | Ca |
what are the main functions of Ca? | bone formation and metabolism, neural transmission and function, initiates skeletal muscel contraction, and maintains cell membrane integrity |
enters through GI tract | Ca |
must have ___ from Ca to be efficient | Vit D |
where is Ca excreted? | to the kidneys and GI tract |
__ and ___ play a big role in the excretion of Ca | PTH and calcitonin |
what is the relationship between Ca and Ph? | inversely proportional= Ca up, Ph down...Ca down, Ph up |
Ca and ___ are directly proportional | Mg.....Mg up, Ca up.....Mg down, Ca down |
what are some main causes of hypocalemia? | < intake, < absorption, > secretion |
what are the main s/s of hypocalemia? | parasthesia, Chvestak's sign, trousseau's sign, > DTR, pathological fracture, skin hair and nail changes, larygospasms, stridor, bruising, bleeding, ECG changes |
what are the ECG changes in hypocalcemia? | prolonged QT interval,dysrythmias |
body doesn't take ___ supplements well | Ca |
high Ca in the blood | hypercalcemia |
what are the main causes of hypercalcemia? | hyperparathyriodism, malignancy, immoblization, renal impairment |
what are the main s/s of hypercalcemia? | anorexia, N/V, abd pain, < bowel sounds, constipation, neuromuscular weakness to flaccidity, < DTR, confusion, depression, lethargy. stupor, coma, renal calculi, ECG changes |
what are the ECG changes for hypercalcemia? | shortened QT interval, inotropic effect, dysrhythmias |
second most abundant ICF cation | Mg |
cofactor for many enzyme activity | Mg |
essential for ATP synthesis, DNA replication and transcription, cellular metabolism, membrane functions, nerve conduction, ion transports, and Ca channel activity | Mg |
ingested through diet | Mg |
how is Mg excreted? | through kidneys |
what are the ECG changes in hypomagnesemia? | prolonged QT interval, dysrhythmias |
low Mg in the blood | hypomagnesemia |
high Mg in the blood | hypermagnesmia |
what are some causes of hypermagnesmia? | > intake, use of Mg antacids and laxatives, renal impairment, endocrine disorders, acidosis |
what are 3 endocrine disorders that may cause hypermagnesmia? | hypoparathyroidism, hypoaldosteroneism, and hypothyroidism |
what are some main s/s of hypermagnesmis? | bradycardia, hypotension, muscle weakness, <DTR, resp. impairment, lethargy |
indirect measure of H ion concentrations | pH |
a substance that can give up an H ion; result of cellular metabolism | Acid |
carbonic acid....exhalable | volatile |
sulfuric, uric acid...excreted in kidneys | nonvolatile |
blood pH < 7.35 | acidosis (acid) |
a substance that can accept an H ion | base |
___ is the primary base in the body | bicarb |
blood pH > 7.45 | alkalosis (base) |
a blood pH between ___ and ___ is incompatible with life | <6.8 and >7.8 |
what are some chemical buffers in the acid-base system? | bicarb-carbonic acid...phosphate system...ammonium...some proteins |
chemical buffers are an ____ system | immediate response |
the most important chemical buffer and is generateed int he kidneys and aids in the elimanation of H | bicarb-carbonic acid |
chemical buffer that aids in excretion of H by the kidneys | phosphate system |
chemical buffer that is added to ammonida in the renal tubules to form ammonium | ammonium |
chemical buffer that aids in buffering ECF | certain proteins |
what are the 3 parts of acid-base regulation? | chemical buffers, respiratory system, and renal system |
regulates teh excretion or retention of carbonic acid | respiratory system |
if pH <, the resp rate and depth ___ | > |
if pH >, the resp rate and depth ___ | < |
fast but weak acid-base regulator | respiratory system (responds within minutes) |
slow but powerful acid-base regulator | renal system (responds within 48 hrs) |
regulates the excretion or retention of bicarb and the excretion of H and nonvolatile acids | renal system |
if pH <, kidneys ____ bicarb | retain |
if pH >, the kidneys ____ bicarb | excrete |
losing bases | metabolic acidosis |
deficit of bicarb | metabolic acidosis |
what are the causes of metabolic acidosis? | ketoacidosis, renal failure, diarrhea |
what are the main s/s of metabolic acidosis? | weakness, tremors, tachypnea, hypothension, confusion, lethargy, dysrhythmias |
what is Kussmaul's? | when pt is panting to get more expiration out (mouth breathing) |
how does the body compensate with metabolic acidosis? | lungs > rate and depth of ventilation...PaCO2 levels <...change is rapid, usually within minutes to hours |
gaining > bases | metabolic alkalosis |
what are the main causes of metabolic alkalosis? | NG suction, K losing thru diuretics |
what are the main s/s of metabolic alkalosis? | bradypnea, parasthsia, confusion, > muscle irritability, tetany, seizures, coma |
what are compenstations for metabolic alkalosis? | lung < rate and depth of ventilation...PaCO2 levels >...change is rapid, usually within minutes to hours |
excessive retention of CO2 | respiratory acidosis |
what are the main causes of resp acidosis? | airway obstruction and hypoventilation |
what are the main early s/s of resp acidosis? | tachycardia, tachypnea, diaphoresis |
what are the late s/s of resp acidosis | bradycardia and hypotension |
what are the compensations for resp acidosis? | kidneys reabsorb more bicarb or excrete more H...bicarb and base excess levels >...change is slow and may take 2-3 days |
excessive elimination of CO2 | respiratory alkalosis |
what is the main cause of respiratory alkalosis? | hyperventilation, anxiety |
what are the main s/s of resp alkalosis? | tachyardia, palpitations, dry mouth, anxeity, profuse perspiration, parasthseia, inability to concentrate |
what are the compensations for resp alkalosis? | kidneys excrete more bicarb...bicarb and base excess levels...change is slow 2-3 days |
what is a tx for resp alkalosis? | breath into paper bag |
kidneys will shut down if ___ | urine is not made |