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Stack #158869

kilgore lvl1: adrenergic antagonists (blockers)

QuestionAnswer
effect of a1-blocker on pupil constriction (Miosis)
effect of a1-blocker on blood vessels of skin & viscera vasodilation
effect of a1-blocker on GI sphincters relaxation
effect of a1-blocker on bladder sphincter relaxation
effect of a2-blocker on presynaptic nerve terminals Release of neurotransmitters (results in SNS stimulation effects)
effect of B1-blocker on Heart decreased rate (neg. chronotrophic) decreased contractility (neg. inotrophic) decreased AV conduction (neg. dromotrophic)
Good way to remember dromotrophic is electrical conduction (singing) "-Dromo- arigato, Mr. Roboto" (Think robots...run on ELECTRICITY) sorry...I am a nerd....
effect of B1-blocker on pancreas increased insulin release (removes blood sugar from system)
effect of B2-blocker on heart, lung & skeletal muscle blood vessels vasoconstriction
effect of B2-blocker on Liver glycogenesis (transformation of glucose to glycogen)
effect of B2-blocker on kidney decreased renin secretion (B/P goes down)
effect of B2-blocker on GI muscles increased motility
effect of B2-blocker on Uterus contraction (no longer in danger...so have the baby already....)
effect of B2-blocker on Bronchial muscles contraction of bronchi
effect of dopamine-blocker on kidney constriction of kidney vasculature
What are some typical side-effects seen with B-adrenergic blockers? hypotention, bradycardia, fatigue depression, hypoglycemia
what happens if a-blockers, B-blockers, CCBs and/or diuretics combine? additive effects resulting in profound hypotension
key facts on phentolamine (Regitine) >a-blocker to reduce PVR and hypertension >can treat pheochromocytoma & establish it's diagnosis >
key facts on prazosin (minipress) a1-blocker >used for hypertension >used for men w/ BPH to relax & dilate vessles and smooth muscle around prostate >big occurence of orthostatic hypotension >take at bedtime
what is a cardioselective B-blocker block B1-receptor stimulation resulting in: >reduced heart rate (neg. chronotrophic) >slowed electrical conduction in heart (neg. dromotrophic) >decreased contractility (neg. inotrophic)
Why do you need to be careful with non-selective B-blockers? they block both B1 and B2 receptors, so they will cause bronchi to contract, which would be bad for someone w/ compromised airways/respiration
key facts on atenolol cardioselective B1-blocker >used for hypertension, dysrhythmias & angina >possesses intrinsic sympathomimetic activity
key facts on esmolol potent short-acting B1-blocker >used for rapid, temp control of ventricular tachydysrhythmias
key facts on propranolo non-selective B-blocker >used for migraine headaches as well as cardiac related uses >NEVER use on client w/ bronchial asthma
key facts on metoprolol B1-blocker >used post-MI to increase survival rate
Created by: gfcfnina
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