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HEMATOLOGIC
| Question | Answer |
|---|---|
| CHEIF FUNCTION | DELIVERY OF SUBSTANCES FOR CELLULAR METABOLISMMICROOORAGNISMS AND INJURY<BR>MAINTENACE OF ACID BASE BALANCE<BR>REMOVE WASIT<BR>DEFENSE AGAINST |
| COMPOSIT. OF BLOOD | 55% PLASMA, 45% BLOOD CELLS |
| THREE TYEPS OF BLOOD CELLS | RBC, PLATELETS, WBC |
| RBC | 4.2-6.2 MIL |
| PLATELETS | 150,000-400,000 |
| WBC | 5000-10,000 |
| ERYTHROCYTES | MOST ABUNDANT<BR>TISSUE OXYGENATION<BR>REVERSE DEFORMITY<BR>120 LIFE |
| LEUKO CYTES | WBC<BR>DEFENDS BODY AND REMOVE DEBRIS |
| GRANULOCYTES | MEMBRAIN BOUND<BR>ENZYMES DESTROY MICROORANISMS<BR>INFLAMMATORY AND IMMUNE FUNCTIONS |
| 4GRANULOCYTES | NEEUTROPHILS<BR>EOSINOPHILS<BR>MAST CELLS<BR>BASOPHILS |
| NEUTRO.. | PMN<BR>PHAGOCYTES (EARLY) |
| EOSINOPHILS | IGE HYPERSENSITIVITY<BR>INCREASE PARASITIC INFECTIONS<BR>INGEST ANTIGEN-ANTIBODY |
| MASTCELLS | CENTRAL CELL<BR>FOUND IN VASCULARAIZED CONNECTIVE TISSUE |
| BASOPHILS | STRUCTURALLY AND FUNCTIONALLY SIMILAR TO MAST CELLS |
| ACTIVATION OF PLATELETS | LEADS TO CONFORMATIONAL CHANGE IN GP RECEPTORS<BR>BINDS TO PROTEINS<BR>SHAPE CHANGES<BR>PSEUDOPODS<BR>ACTIVATION OF ARACHIDONIC PATHWAY |
| ADHESION | LOSS OF ENDO CELLS |
| AGGREGATION | RELEASE OF TXA<BR>HEPARIN ENHANCES CLOT FORMATION<BR>PLUG IS TABLE<BR>ACTIVATES COAGULATION |
| ANEMIA | DECREASE RBC MASS |
| WORKING DEF OF ANEMIA | DECREASED RBC COUNT HEMOG HEMATO |
| MECHANISM OF ANEMIA <BR>PRODUCTION DISORDER | IRON<BR>V-B12<BR>FOLATE<BR>THALASSEMIAS |
| HEMATOPOIETIC CELL FAILURE | APASTIC, MAY BE DUE TO DRUGS, RADIATION, INFECTIONS, TOXINS |
| INCREAED CELL LOSS | MEHOLYTIC SICKLE CELL, THALASSEMIAS |
| SICKLE CELL ANEMIA | HEREDITARY<BR>HEMOGLOBINOPATHY WITH PARTIAL OR COMPLETE REPLACEMENT OF NORMAL HEMOGLOBIN WITH HEMOG S<BR>S ASSUMES A SICKLED SHAPE AND DERMS RBCS<BR>HEMOLYTIC ANEMIA RESULTS |
| NORMAL HEMOGLOBIN | DISC SHAPED<BR>SOF<BR>EASILY FLOW THROUGH BV<BR>LIVES FOR 120 DAYS |
| SICKLE | SICLES SHAPED<BR>HARD<BR>OFTEN GET STUCK IN SMALL BLOOD VESSELS<BR>LIVES FOR 20 DAYS |
| POLYCYTHEMIA 3 TYPES | VERA, SECONDARY, RELATIVE |
| VERA | NEOPLASTIC TRANSFORMATION OF HEMATOPOETIC STEM CELLS |
| SECONDARY | CHRONIC HYPOXEMIA |
| RELATIVE | DEHYDRATION |
| POLYCYTHEMIA RESULTS IS | INCREASED BLOOD VISCOSITY AND RISK OF THROMBI |
| TREATMENT OF POLYCYTHEMIA | PHLEBOTOMY, BONE MARROW SUPPRESSANTS |
| INTRINSCI PATHWAY | ACTIVATED WHEN FACTOR XII CONTACTS SUBENDOTHELIAL SUBSTANCES EXPOSED BY VASCULAR INJURY |
| EXTRINSIC PATHWAY | ACTIVATED WHEN TISSUE FACTOR IS RELEASED BY DAMAGED ENDOTHELIAL CELLS |
| TESTS OF BONE MARROW FUNCTION | ASPIRATION<BR>BIOPSY<BR>MEASUREMENT OF BONE MARROW IRON STORES<BR>DIFFERENTIAL CELL COUNT |
Created by:
leah_76
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