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NUR1213_Exam 4
OXYGENATION: PERFUSION (ALTERED TISSUE PERFUSION) SHOCK
| Question | Answer |
|---|---|
| IN THE COMPENSATORY STAGE, THE B/P.. | REMAINS WITHIN NORMAL LIMITS |
| S&S OF PT IN COMPENSATORY STAGE? | COOL & CLAMMY SKIN, HYPOACTIVE BOWEL SOUNDS, DECREASE IN URINE OUTPUT |
| THE CLINICAL MANIFESTATIONS OF COMPENSATORY STAGE RESULT IN? | METABOLIC ACIDOSIS (RR INCREASES) |
| COMPENSATORY STAGE -> METABOLIC ACIDOSIS -> RAPID RESPIRATORY RATE = REMOVAL OF EXCESS C02 BUT RAISES BLD pH OFTEN CAUSING.. | COMPENSATORY RESPIRATORY ALKALOSIS |
| ALKALOTIC STATE CAUSES? | MENTAL STATUS CHANGE (ie: CONFUION/COMBATIVENESS), & ARTERIOL DIALATION. |
| MEDICAL MANAGEMENT DURING THE COMPENSATORY STAGE OF SHOCK | FLUID REPLACEMENT & MEDICATION THERAPY (INITIATED TO MAINTAIN ADEQUATE B/P/REESTABLISH& MAINTAIN ADEQUATE TISS PURFUSION.) |
| CLINICAL FINDINGS DURING COMPENSATORY STAGE.. | <CENTER>B/P NORMAL</CENTER><CENTER>HRT RATE >100 BPM </CENTER><CENTER>>20 BREATHS/MIN </CENTER><CENTER>COLD/CLAMY SKIN </CENTER><CENTER> DECREASED URINE OUTPUT </CENTER><CENTER>CONFUSION </CENTER><CENTER>REPIRATORY ALKALOSIS </CENTER> |
| CLINICAL FINDINGS DURING PROGRESSIVE STAGE.. | <CENTER>SYS BP <80-90MM Hg </CENTER><CENTER>HEART RATE >150 BPM </CENTER><CENTER>RAPID/SHALLOW RESPIRATIONS; CRACKLES </CENTER><CENTER>MOTTLED, PETECHIAE SKIN </CENTER><CENTER>URINE OUTPUT 0.5 ML/KG/H </CENTER><CENTER> LETHARGY</CENTER><CENTER> METABOLIC |
| CLINICAL FINDINGS DURING IRREVERSABLE STAGE.. | <CENTER>BP-REQUIRES MECHANICAL OR RX SUPPORT </CENTER><CENTER>ERRATIC OR ASYSTOLE HEART RATE </CENTER><CENTER>REQUIRES INTUBATION (RESP. STATUS) </CENTER><CENTER> JAUNDICE</CENTER><CENTER> URINE OUTPUT= ANURIC, REQUIRES DIALYSIS</CENTER><CENTER> UNCONSCIO |
| MEAN ARTIRAL BP= | CARDIAC OUTPUT x PERIPHERAL RESISTANCE |
| CARDIAC OUTPUT IS DETERMINES BY? | STROKE VOLUME (AMOUNT BLD EJECTED @ SYSTOLE)& HR |
| TISSUE PERFUSION & ORGAN PERFUSION DEPEND ON? | MAP |
| MAP MUST EXCEED ____ mm Hg FOR CELLS TO RECIEVE O2 & NUTRIENTS | 65 mm Hg |
| HYPOVOLEMIC SHOCK.. | OCCURS WHEN THERE IS A DEREASE IN THE INTRAVASCULAR VLM |
| CARDIOGENIC SHOCK... | OCCURES WHEN THE HEART HAS A IMPAIRED PUMPING ABILITY; MAY BE OF CORONARYOR NON CORONARY EVENT ORIGIN |
| SEPTIC SHOCK.. | CAUSED BY INFECTION |
| ANAPHYLACTIC SHOCK.. | CAUSED BY A HYPERSENSITIVITY REACTION |
| SHOCK.. | PHYSIOLOGICAL STATE IN WHICH THERE IS INADEQUATE BLOOD FLOOR & NUTRIENTS DELIVERED TO THE CELLS AND TISSUES OF THE BODY. |
| COLLOIDS ARE.. | INTRAVENOUS SOLUTIONS THAT CONTAIN MOLECULES THAT ARE TOO LARGE TO PASS THRU CAPILLARY MEMBRANES |
| CRSTALLOIDS ARE.. | ELECTROLYTE SOLUTIONS THAT MOVE FREELY BTWN THE INTRAVASCULAR COMPARTMENT AND INTERSTITIAL SPACES |
| CIRCULATORY SHOCK.. | SHOCK STATE RESULTING FRM DISPLACEMENT OF BLD VLM CREATING A RELATIVE HYPOVOLEMIA & INADEQUATE DELIVERY OF O2 TO THE CELLS (AKA DISTRIBUTIVE SHOCK) |
| PHYSIOLOGICAL RESPONSES OF ALL TYPES OF SHOCK | <CENTER>HYPOPERFUSION OF TISSUE</CENTER><CENTER>HYPERMETABOLISM </CENTER><CENTER>ACTIVATION OF INFLAMMATORY RESPONSES </CENTER> |
| BODIES RESPONSES TO SHOCK | <CENTER>CTIVATING SYMPATHETIC NERVOUS SYS </CENTER><CENTER>MOUNTING HYPERMETABOLIC </CENTER><CENTER> & INFLAMMATORY RESPONSE</CENTER> |
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