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glomerualr disease
Stack #172434
| Question | Answer |
|---|---|
| Injury to the glomerulus is often reflected by the presence of what two things in the urine | blood (hematuria) or protein (proteinuria) |
| type of glomerluar disease, This is characterized by hematuria, red blood cell casts, azotemia, hypertension, and oliguria | NEPHRITIC SYNDROME |
| In general, these disorders are immunologically mediated. | NEPHRITIC SYNDROME |
| This may occur at any age but is most commonly seen in children 1-3 weeks after a Group A ß-hemolytic streptococcal infection (usually strep throat) | ACUTE PROLIFERATIVE (POST-INFECTIOUS) GLOMERULONEPHRITIS |
| Acute proiferative lomerulonephritis is due to | antigen-antibody complexes (Type III immune hypersensitivity reaction) getting trapped in glomeruli. These complexes damage the basement membranes of the glomerular capillaries |
| what is the endresult of the basement membranes ofthe glomerular capillariesbeing damaged with acue proliferatie glomerulonephritis? | blood and protein to escape into the urine collecting system. |
| Symptoms of what type of glomerular disease? In children, there is malaise, fever, oliguria, hematuria, nausea, periorbital edema, and mild-moderate hypertension. | NEPHRITIC SYNDROME |
| What presents in the urine of a child ith acute proliferative glomerulonephriti? | Red blood cell casts (red blood cells trapped in a protein matrix) and mild amounts of protein (< 1 gm) are present in the urine. |
| What percent of children recover fully while a few may progress to chronic glomerulonephritis. | 95 |
| The prognosis in adults with acute proliferatie glomerulonephritis is a little poorer with about recovery. | 60 |
| This is a clinicopathologic syndrome in which there is a rapid progression to renal failure | RAPIDLY PROGRESSIVE GLOMERULONEPHRITIS (RPGN) - |
| what is the length from onset to renal failure in clients with rapidly progressive gloerulonephritis? | weeks |
| The following are symptoms of what type of glomeulonephrtis?his may have an abrupt onset of oliguria and hematuria with lesser degrees of hypertension, edema, and proteinuria. | rapidly progressive |
| This is a constellation of signs characterized by proteinuria (> 3.5 gm/day), hypoalbuminemia (reversed albumin:globulin ratio), hyperlipidemia (increased LDL and/or VLDL), lipiduria (free fat and oval fat bodies in urine), and edema (pitting edema mos | NEPHROTIC SYNDROME |
| proteinuria, what is numerical value | (> 3.5 gm/day), |
| Complications of nephrotic syndrome include | infections (due to loss of immunoglobulins and complement) and thrombosis (due to loss of anticoagulant factors) |
| The major diseases causing the nephrotic syndrome are: | MINIMAL CHANGE DISEASE (lipoid nephrosis, nil disease),MEMBRANOUS GLOMERULONEPHROPATHY |
| This is the most common cause of nephrotic syndrome in children, but it can occur in adults as well. | MINIMAL CHANGE DISEASE (lipoid nephrosis, nil disease) - |
| Children with minimal change disease respond to what type of therapy? | steroid |
| This is the most common cause of nephrotic syndrome in adults (usually young to middle aged) | MEMBRANOUS GLOMERULONEPHROPATHY - |
| type ofnephrotic syndrome, characterized by thickening of the glomerular capillary basement membranes. | memranous glomerulonephopathy |
| What are three symptoms f membanous glomerlonephropathy? | develop increasing blood urea nitrogen (BUN), hypertension, and progressive renal failure. |
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