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Local Anesthesia
WREB Anesthesia Study Guide
Question | Answer |
---|---|
Allergic reaction management; immediate vs delayed | epi if immediate, benadryl if delayed |
Amides are metabolized by what? | liver (prilocaine is in the lungs) |
Amides are excreted by what? | kidneys |
Asthma patients are sensitive to what additive? | sulfites (no epi) |
Avoid esters and articaine if.... | pt.'s have atypical pseudocholinesterase, sulfur allergy, and myasthinia gravis |
Brand name for lidocaine | xylocaine |
brand name for mepivicaine | polocaine |
brand name for mepivicaine plain | carbocaine |
brand name for articaine | septocaine |
brand name for bupivicaine | marcaine |
brand name for prilocaine | citanest |
difference between block and infiltration | block anesthetic is deposited close to main nerve trunk and covers a larger area,filtration is deposited near large terminal nerve endings |
Dose of AMSA | .9-1.8 ml |
Dose of ASA | .9-1.2ml |
Dose of GP | .4-.6ml |
Dose of IA | 1.5-1.8ml |
Dose of infiltrations | .6 ml |
Dose of LB | .2-.3ml |
Dose of MI | .6ml |
Dose of MSA | .9-1.2ml |
Dose of NP | .4ml |
Dose of PSA | .9-1.8ml |
Esters and articaine are metabolized by what? | pseudocholinesterase in blood plasma |
Esters and articaine are excreted by what? | kidneys |
First sign of local anesthesia toxicity | CNS excitment |
How far do you insert the needle into the IANB? | 20-25 mm |
How much epi is in lidocaine 1:50,000 | .036mg |
How much epi is in lidocaine 1:100,000 | .018mg |
How much epi is in 1:200,000 | .009 mg |
How much LA in one carp of 2% lido? | 36 mg |
How much LA in one carp of 4% articaine | 72 mg |
How much LA in one carp of 3% mepivicaine | 54mg |
How much LA in one carp of .5% bupivicaine | 9mg |
How much of the nerve must be bathed in LA to achieve anesthesia | 8-10mm |
How would you best achieve hemostasis | 1:50,000 |
If a patient is allergic to red wine, what LA would you give them? | anything plain, no vaso |
If there is methemoblobinemia | avoid prilocaine and benzocaine |
If they are taking beta blockers | limit or avoid vaso's |
If they are taking phenothizides | do not use 1:50,000 |
if they are taking tricyclic antidepressants | no levo |
injection and landmarks for buccal tissue of mandibular molars | LB mucous membrane distal and lateral to most posterior molar |
Injection and landmarks for maxillary incisors | ASA, height of the mucobuccal fold about the 1st premolar |
LA overdose leads to | CNS excitation, CNS depression, Coma, convulsions, respiratory arrest |
Management of a hematoma | cold compression and pressure to bleeding site for 3-5 min |
Management of tismus | heat and jaw excersice |
maxillary division of trigeminal nerve is | sensory only |
The metabolic by-product responsible for allergic reactions to the ester classification of anesthetic is | PABA |
Molar root does not anesthetized by the PSA | mesiobuccal root of first premolar |
Most common LA without a Vaso | 3% mepivicaine |
Nerve fiber that is lightly mylinated and causes sharp pain | A |
Nerve fiber without myelin that causes dull achy pain | C |
Nerve impulses are spread by | saltatory conductions |
Nerve that innervates the soft palate | lesser palatine |
on which oral tissue is topical anesthetic least effective | keratinized |
a patient complains of a lesion on the lower lip a day after LA administration, what is it most likely | self mutulation |
purpose of the myelin sheath | protective covering for the nerves |
smaller needle gauge equals | larger |
Tachyphylaxis | the rapid decreased in the response to a drug after repeated doses over a short period of time |
volume in one carpule of LA | 1.8 ml |
what are the most likely causes of an overdose from a local anesthetic | solution was administered too rapidly or solutions was inadvertently injected intravasculary |
what causes psychogenic pain | mental or emotional problems that affect the experience of pain |
what distributes the incoming signals to the CNS | axon |
what to beta blockers do? | decrease BP |
what does a yellow brown tint in the carpule indicate | oxidation |
what does LA do to depolarization | inhibit the Na influx by displacing the Ca ions |
what does the alpha adrenergic receptors do? | smooth muscle contraction in arterioles and veins |
what does the beta adrenergic receptors do? | cardiac stimulation then smooth muscle relaxation |
what exits the foramen ovale | mandibular nerve |
what exits the foramen rotundum | maxillary nerve |
what exits the incisive foramen | nasopalatine nerve |
what exits the mandibular foramen | inferior alveolar nerve |
what happens during depolarization | slow Na ion influx until firing threshold is reached |
What happens during rapid depolarization | once firing threshold has been reached there is a rapid influx of sodium ions |
what happens during repolarization | Na begins to move with concentration gradient out of the cell, there is an absolute refractory period. Na continues to move out of cell with help of sodium pump, there is a relative refractory period until resting state is reached |
What initiates nerve conduction | dendrites |
what is responsible for the duration of action to LA | protein binding |
What is the function of sodium bisulfite to LA solutions | antioxidant that prevents biodegradiation |
What is the function of sodium chloride in LA | isotonicity |
What is the cardiac dose of epi | 0.04 |
what is the site of action for a LA | nerve membrane |
What sensation is lost first | pain |
Which anesthetic affects the CVS first | bupivicaine |
what anesthetics affect the CNS | all besides bupivicaine |
which anesthetics are category B | lido, prilo others are C |
Which anesthetics have a weak vasodilation | mepivicaine and prilocaine |
which is true of topical anesthetics | no systemic issues |
why do you use vaso | increases duration and depth of LA |
why would there be mild burning during administration of anesthetics | ph of solution, too warm, contamination |
Why does infiltration work better on the maxilla as opposed to the mandible | mandible has denser bone. |