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dysrythmias
antidysrythmias
| Question | Answer |
|---|---|
| Dysrhythmias | Abnormalities of electrical conduction or rhythm in heart. A defect of electrical impulses across myocardium and can occur in both healthy and heart may decreased cardiac output. Persistent or severe this with increased risk of stroke and heart. |
| Symptoms of dysrhythmias | Dizziness and weakness decrease exercise tolerance SLB painting palpitations or sensation that their heart has skipped a beat |
| Diseases/conditions associated with dysrhythmias | HTN cardiac valve disease such as aortic stenosis coronary artery disease medications such as digoxin low potassium or magnesium levels myocardial infarction stroke diabetes mellitus heart failure |
| Cardioversion | electrical shocks to your heart through electrodes placed on your chest to restore normal heart rhytm |
| defibrillation | stopping of fibrillation of the heart by administering a controlled electric shock to restore normal rhythm |
| Fibrillation | Complete disorganization of rhythm |
| Sinus rhythm | Action potential approximately 75 times per minute under resting conditions with a normal range of 60 to 100 bpm |
| Electrocardiogram (ECG) | Graphic recording device to measure the way of electrical activity across the myocardium. Used to diagnose many types of heart conditions. Changes to wave patterns or timing reveal certain pathologies |
| Nonpharmacological therapies for dysrhythmias | Cardioversion/defibrillation- electrical shock to allow us a note to regain control catheter ablation- identify/destroy apparent cardiac cells cardiac pacemaker- paces heart at set rate implantable cardioverter defibrillator- pacemaker/defibrillator |
| Anti-dysrhythmic drugs | Act by altering specific electrophysiological properties of the heart by blocking flow through ion channels (conduction) or altering autonomic activity (automaticity) |
| Antidysrhythmic drug classes | Class I: sodium channel blockers class II: beta-adrenergic antagonists class III: potassium channel blockers class for calcium channel blockers |
| procainamide | Class: sodium channel blockers block sodium ion channels and suppress the topic activity to correct atrial and ventricle dysrhythmias. Action: class IA antidysrhythmic Admin: supine position IV admin due to severe hypotension. Preg Cat C |
| procainamide side/contraindictions | side: N&V abdominal pain hypotension H/A confusion psychosis Con: patients with complete AV block severe HF myasthenia gravis blood dyscrasias |
| procainamide warning/interactions | warning: increased tire of anti-nuclear antibodies lupus like syndrome reserved for life-threatening dysrhythmias new/worsen dysrhythmias agranulocytosis bone marrow depression neutropenia hypoplastic anemia thrombocytopenia d/c if blood dyscrasias |
| procainamide interactions | Drug: additive cardiac the present effects if administered with other anti-dysrhythmias anticholinergic side effects will occur if given lapse:^ AST, ALT, ALP, LDH, serum bilirubin. False positive Coombs test & ANA titers may occur |
| amiodarone/ Cordarone | Class: potassium channel blockers delay repolarization in less than refractory period stabilizes dysrhythmias. To treat resistant ventricular tachycardia atrial dysrhythmias with heart failure. Action: Class III anti-dysrhythmic |
| amiodarone/ Cordarone admin/side | Admin: hypokalemia and hypomagnesemia corrected prior to therapy Preg Cat D Side: pulmonary toxicity elevated liver enzymes thyroid dysfunction bluegray scan word vision rashes photosensitivity anorexia fatigue dizziness hypotension N&V |
| amiodarone/ Cordarone warning | Warning: (oral form only) pneumonia like syndrome in the lungs assessment of lung function amiodarone has probed this rhythmic action and calls bradycardia cardiogenic shock or AV block. Mild liver injury. |
| amiodarone/Cordarone conraindictions/labs | con: Bradycardia cardiogenic shock sick sinus syndrome severe sinus node dysfunction third-degree AV block Labs: ^ levels of ANA ALT AST ALP TSH T4 |
| amiodarone/Cordarone interactions-drug/food | Inter: ^ serum digoxin levels block metabolism of warfarin requires lower dose of anticoagulant beta blocker/CCB worsen sinus bradycardia sinus arrest or AV block ^ phenytoin levels herbal: echinacea ^ hepatotoxicity aloe ^ effect of med |
| lifespan of antidysrhythmics | As patients metabolize antidysrhythmics differently monitor ethnically diverse patients frequently to ensure optimal therapeutic effects and to minimize adverse effects older adults hypotension |
| pt teaching for antidysrhythmics | Record peripheral pulse 1 min before taking abnormal call dr BP if below 90/60 in different positions take first dose at bedtime pulmonary toxicity signs of HF report immediately report vision changes wear protective clothing dr more 1 dose missed |
Created by:
bryant-41892
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