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Med Surg 2 Final

QuestionAnswer
end product of protein metabolism urea
what electrolyte imbalance is a major issue with renal failure? hyperkalemia
what does BUN stand for, and what is the normal range? BUN=blood, urea, nitrogen; normal range 10-20
normal creatinine range? 0.6-1.3
what does erythropoietin do? stimulates production of RBCs
a condition in which the kidneys are unable to remove accumulated metabolites from the blood, resulting in altered fluid, electrolyte, and acid/base balance; results from many different causes renal failure
a progressive, irreversible deterioration of renal function that results in azotemia; occur for 3 months or longer chronic renal failure
rapid loss of renal function d/t damage to the kidneys; depending on the duration and severity, a wide range of potentially life-threatening metabolic complications can occur, including metabolic acidosis as well as fluid and electrolyte imbalances acute renal failure
some contributing factors of ____ are: -hypovolemia -hypotension -reduced cardiac output and heart failure -obstruction of kidney or lower urinary tract by tumor, blood clot, or kidney stone -bilateral obstruction of the renal arteries or veins AKI
occurs in 60-70% of cases; result of impaired blood flow that leads to hypoperfusion of the kidney Prerenal AKI
commonly caused by volume depletion (burns, hemorrhage, GI losses), hypotension (sepsis, shock), and renal artery stenosis, ultimately leading to a decrease in the GFR Prerenal AKI
result of actual parenchymal damage to the glomeruli or kidney tubules; commonly caused by prolonged renal ischemia, nephrotoxic agents, infectious processes Intrarenal AKI
most common type of intrinsic AKI; results from the destruction of tubular epithelial cells w/abrupt decline in renal function; CKD, DM, heart failure, HTN and cirrhosis can lead to this Acute tubular necrosis (ATN), or AKI in which there is damage to the kidney tubules
intratubular obstruction, tubular back leak (abnormal reabsorption of filtrate and decreased urine flow through tubule), vasoconstriction, and changes in glomerular permeability--> decreased GFR, progressive azotemia, & fluid/electrolyte imbalances Acute tubular necrosis (ATN)
usually results from obstruction distal to the kidney by conditions such as renal calculi, strictures, blood clots, BPH, malignancies, and pregnancy; pressure rises in the kidney tubules, and eventually the GFR decreases Postrenal AKI
phase of AKI: begins with the initial insult and ends when oliguria develops initiation period
phase of AKI: increase in serum concentration of substances usually excreted by the kidneys (urea, creatinine, uric acid, organic acids, and intracellular cations [K+ and Mg]; uremic Sx first appear and life-threatening conditions (hyperkalemia) develop oliguria period
phase of AKI: marked by a gradual increase in urine output, which signals that glomerular filtration has started to recover; lab values stabilize and eventually decrease; watch for a decrease in Na, K, and dehydration (Prerenal AKI can occur) diuresis period
phase of AKI: signals the improvement of renal function and may take 3-12 months; lab values return to pt’s normal level; although a permanent 1-3% reduction in the GFR may occur, it is not clinically significant recovery period
The minimum amount of urine needed to rid the body of normal metabolic waste products is? 0.5 mL/kg/hr
hypoperfusion; elevated BUN, creatinine, urine specific gravity; decreased urine output, urine sodium; urine sediment normal to few casts Prerenal AKI
parenchymal damage; elevated BUN, creatinine, urine sodium; urine output varies (often low); increased urine sodium; abnormal casts and debris; urine specific gravity low normal Intrarenal AKI
obstruction; elevated BUN, creatinine; urine output varies (may be low or sudden anuria); urine sodium varies; usually normal sediment; urine specific gravity varies Postrenal AKI
diagnosing AKI: labs? radiology? -Labs: UA, serum BUN and creatinine, serum electrolytes, CBC -Diagnostic: ***renal US***, CT of kidneys, IV pyelogram, renal biopsy
Tx: prevention (maintain adequate hydration and diuresis); perform rapid interventions and and correct the cause; dialysis; manage systemic problems (infection, electrolyte imbalances); diet: high calorie, low protein/potassium/phosphorus) AKI
pharmacology in AKI -IV fluids -diuretics (loop or osmotic) -antihypertensives -Tx of increased serum potassium (Kayexalate) -electrolyte replacement -phosphorus-binding agent (calcium); give with meals
hyperkalemia EKG changes tall, tented, or peaked T waves
Sx: irritability, abd cramping, diarrhea, paresthesia, and generalized muscle weakness (may present as slurred speech, difficulty breathing, paresthesia, and paralysis), both cardiac and other muscular function declines-->medical emergency hyperkalemia
Many meds are eliminated through the kidneys; therefore, dosages must be reduced when a patient has AKI. Examples of commonly used agents that require adjustment? -ATBs (especially aminoglycosides) -digoxin (Lanoxin) -phenytoin (Dilantin) -ACE inhibitors -magnesium-containing agents
nursing management of AKI -fluid balance (daily weights, strict I&Os) -electrolyte imbalance -activity intolerance -skin care -pt knowledge of disease and managment -nutritional status -psychological support -prevention of infection and other complications
an umbrella term that describes kidney damage or a decrease in the GFR lasting for 3 or more months; associated with decreased quality of life, increased health care expenditures, and premature death Chronic Kidney Disease (CKD)
stage of CKD: kidney damage with normal or increased GFR (≥90 mL/min/1.73 m2) stage 1
stage of CKD: mild decrease in GFR (60–89 mL/min/1.73 m2) stage 2
stage of CKD: moderate decrease in GFR (30–59 mL/min/1.73 m2) stage 3
stage of CKD: severe decrease in GFR (15–29 mL/min/1.73 m2) stage 4
stage of CKD: end-stage kidney disease or chronic kidney disease (GFR <15 mL/min/1.73 m2) stage 5
causes: DM (primary cause) HTN (second leading cause) chronic glomerulonephritis pyelonephritis or other infections obstruction of urinary tract hereditary lesions vascular disorders nephrotoxic meds or other toxic agents CKD
Patients with CKD are at increased risk for? cardiovascular disease---leading cause of morbidity and mortality
Treatment of HTN, anemia, and hyperglycemia and detection of proteinuria all help to slow disease progression and improve patient outcomes CKD
elevated serum creatinine levels (indicate underlying kidney disease); anemia (d/t decreased erythropoietin production by kidney); metabolic acidosis; abnormalities in Ca and P; fluid retention CKD
normal GFR? 90
urine lab tests (specific gravity, protein, blood, casts); urine culture (infection); urine creatinine clearance (evaluates GFR & renal function); serum BUN (identifies severity of azotemia); serum creatinine level (identifies renal impairment) CKD
neurologic effects of ESRD Asterixis Behavior changes Burning of soles of feet Confusion Disorientation Inability to concentrate Restlessness of legs Seizures Tremors Weakness and fatigue
integumentary effects of ESRD Coarse, thinning hair Dry, flaky skin Ecchymosis Gray-bronze skin color Pruritus Purpura Thin, brittle nails
CV effects of ESRD Engorged neck veins Hyperkalemia Hyperlipidemia Hypertension Pericardial effusion Pericardial friction rub Pericardial tamponade Pericarditis Periorbital edema Pitting edema (feet, hands, sacrum)
pulmonary effects of ESRD Crackles Depressed cough reflex Kussmaul-type respirations Pleuritic pain Shortness of breath Tachypnea Thick, tenacious sputum Uremic pneumonitis
GI effects of ESRD Ammonia odor to breath (“uremic fetor”) Anorexia, nausea, and vomiting Bleeding from gastrointestinal tract Constipation or diarrhea Hiccups Metallic taste Mouth ulcerations and bleeding
hematologic effects of ESRD Anemia Thrombocytopenia
reproductive effects of ESRD Amenorrhea Decreased libido Infertility Testicular atrophy
musculoskeletal effects of ESRD Bone fractures Bone pain Footdrop Loss of muscle strength Muscle cramps Renal osteodystrophy
occurs in ESKD b/c kidneys are unable to excrete increased loads of acid; decreased acid secretion results from inability of kidney tubules to excrete ammonia and to reabsorb sodium bicarb; also decreased excretion of phosphates and other organic acids metabolic acidosis
medication precautions of ESRD -meds excreted by kidneys will increase half life and plasma levels -caution w/giving protein-bound meds (low plasma protein levels can result in toxicity) -avoid/use extreme caution w/nephrotoxic meds -if on hemodialysis--will med be dialyzed?
Is it ok to give cardiac meds right before dialysis? no
pharmacologic management in ESRD -calcium supplements -phosphorus binders -antihypertensives and CV agents -erythropoietin -anticonvulsants (if necessary)
ESRD diet -restriction of protein, fluids, sodium, food high in potassium and phosphorus -high calorie (carbs and fats) -vitamin supplication -(high protein if on dialysis)
nursing interventions for assessing fluid status -daily weight -I&O balance -skin turgor and presence of edema -JVD -B/P, pulse rate and rhythm -respiratory rate and effort
potential sources of fluid -meds and fluids used to take or administer medications: oral and IV -foods
problems/complications of ESRD -hyperkalemia -pericarditis, pericardial effusion, pericardial tamponade -HTN -anemia -bone disease and metastatic calcifications
About 30–50% of patients with CKD develop _____ due to uremia; fever, chest pain, and a pericardial friction rub are classic signs. pericarditis
prevents death but does not cure kidney disease and does not compensate for the loss of endocrine or metabolic activities of the kidneys hemodialysis
how long do you have to wait to use a long-term access catheter for hemodialysis? 2-3 months; needs time to "mature"
access catheter for hemodialysis: assess fistula for audible ____ and palpate for ____ (audible) bruit, (palpate) thrill
two big complications of hemodialysis infection (local or systemic), injury to fistula site
goals are to remove toxic substances and metabolic wastes and to reestablish normal fluid and electrolyte balance peritoneal dialysis
monitor/assess what when patient on dialysis? -vascular access site -S/Sx of infection -fluid balance -S/Sx of uremia and electrolyte balance (check labs--BUN, creatinine, BMP/CMP) -cardiac/resp status
a type of kidney disease with glomerular inflammation; S/Sx: hematuria, edema, azotemia, proteinuria, HTN; may be mild or may progress to AKI or death; Tx: supportive care, diet, ATBs, steroids, immunosuppressants acute nephritic syndrome
prolonged exposure to an environmental temperature >39.2°C (102.5°F); as seen in elderly being outside too long or children/pets in hot cars nonexertional heat stroke
caused by strenuous activity that occurs in hot environments; as seen in farmers or marathon runners exertional heat stroke
people not acclimated to heat, older or very young people, ill or debilitated people, and persons taking some medications are at high risk heat stroke (aka hyperthermia)
manifestations: CNS dysfunction, elevated temp of 40.6°C (105°F) or higher, hot dry skin, anhidrosis (no sweating), tachypnea, hypotension, and tachycardia heat stroke (aka hyperthermia)
tachypnea, hypotension, and tachycardia can indicate what? shock
-ABCs; reduce temp to 39.2°C (102°F) as quickly as possible -cooling methods -monitor temp, VS, ECG, CVP, LOC, I&O -IVs--replace fluid losses -meds: anticonvulsant, K+, Na bicarb, benzos management of a pt with heat stroke (hyperthermia)
-trauma from exposure to freezing temp and freezing of fluid in the intracellular/intercellular spaces -S/Sx: hard, cold, and insensitive to touch; may appear white or mottled, may turn red and painful as rewarmed frostbite
-extent of injury is not always initially known -controlled but rapid rewarming; 37° to 40°C circulating bath for 30- to 40-minute intervals -analgesics for pain -do not massage or handle; if feet are involved, do not walk frostbite
-internal core temp is 35°C or less -older adults, infants, persons with concurrent illness, homeless people, and trauma victims are at risk -ETOH ingestion increases susceptibility -physiologic changes in all organ systems -monitor continuously hypothermia
-use ABCs, remove wet clothing, rewarm, supportive care -active core rewarming (cardiopulmonary bypass, warm fluid administration, warm humidified O2, warm peritoneal lavage) -passive external rewarming (warm blankets and over the bed heaters) management of a pt with hypothermia
cold blood returning from the extremities has high levels of ___ ___ and can cause potential cardiac dysrhythmias and electrolyte disturbances lactic acid
-comprehensive symptom management, psychosocial care, and spiritual support needed to **enhance the quality of life** for any person with advanced illness -interdisciplinary collaboration -settings: hospitals, SNFs, outpatient palliative care
coordinated program of interdisciplinary services provided by professional caregivers and trained volunteers to patients with **serious, progressive illnesses that are not responsive to cure** hospice care
area where breast cancer usually found upper outer quadrant
In general, the lesions are nontender, fixed rather than mobile, and hard with irregular borders. breast cancer
advanced signs of breast cancer skin dimpling nipple retraction skin ulceration
removal of the breast tissue, nipple–areola complex, and a portion of the axillary lymph nodes (ALND); used for invasive breast cancer modified radical mastectomy
removal of the breast and nipple–areola complex but does not include ALND; used for non-invasive breast cancer total mastectomy
surgery/Tx to remove a breast tumor and a margin of tissue around the tumor without removing any other part of the breast; may or may not include lymph node removal and radiation therapy breast conservation treatment
biopsy of first node (or nodes) in the lymphatic basin that receives drainage from the primary tumor in the breast; less invasive alternative to ALND and is considered a standard of care for the treatment of early-stage breast cancer sentinel lymph node biopsy
chronic swelling of an extremity due to interrupted lymphatic circulation, typically from an axillary lymph node dissection lymphedema
a solid swelling of clotted blood within the tissues hematoma
a pocket of clear serous fluid that sometimes develops in the body after surgery seroma
method of breast reconstruction in which a flap of skin, fat, and muscle from the lower abdomen, with its attached blood supply, is rotated to the mastectomy site transverse rectus abdominal myocutaneous (TRAM) flap
-well-differentiated cells resemble normal cells of the tissue from which the tumor originated -tumor grows by expansion and does not infiltrate surrounding tissues; usually encapsulated -rate of growth is usually slow -does not spread by metastasis benign
-cells undifferentiated and may bear little resemblance to cells of original tissue -grows at periphery and overcomes contact inhibition to invade and infiltrate surrounding tissues -rate of growth variable and depends on level of differentiation malignant
spread of cancer cells from the primary tumor to distant sites; abnormal cells that have invasive characteristics; infiltrate other tissues; spreads 3 ways: lymphatic, hemotogenous, angiogenesis metastasis
performed to obtain a tissue sample for histologic analysis of cells suspected to be malignant diagnostic surgery, or biopsy
used for small, easily accessible tumors of the skin, upper or lower gastrointestinal and upper respiratory tracts excisional biopsy
performed if tumor mass is too large to be removed; a wedge of tissue from tumor is removed for analysis incisional biopsy
performed to sample suspicious masses that are easily and safely accessible, such as some masses in the breasts, thyroid, lung, liver, and kidney needle biopsy
often performed on an outpatient basis; is warranted when the mass is small; includes removal of the mass and a small margin of normal tissue that is easily accessible local excision
removal of the primary tumor, lymph nodes, adjacent involved structures, and surrounding tissues that may be at high risk for tumor spread wide (or radical) excision
removing non-vital tissues or organs that are at increased risk of developing cancer; ex-getting a mastectomy d/t family history of breast cancer prophylactic (or risk reduction) surgery
performed in an attempt to relieve symptoms, such as ulceration, obstruction, hemorrhage, pain, and malignant effusions; ex-PEG tube insertion for enteral nutrition, colostomy for bowel obstruction palliative surgery
may follow curative or extensive surgery in an attempt to improve function or obtain a more desirable cosmetic effect reconstructive surgery
most common type of radiation therapy used for cancer treatment; a machine is used to aim high-energy rays from outside the body into the tumor external beam radiation therapy (ERBT); aka teletherapy
radiation therapy in which a small container of radioactive material is implanted in the body, in or near the cancerous tumor internal radiation (aka brachytherapy)
agents used to destroy tumor cells by interfering with cellular function, including replication and DNA repair chemotherapy
1-inflammation/irritation of the mucous membranes in the mouth (gums, tongue, lips); 2-a complication of some cancer therapies in which the lining of the digestive system becomes inflamed (often seen as sores in the mouth) 1-stomatitis, 2-mucositis
nursing interventions for maintaining fluid and electrolytes -Intake and output, daily weights -Assess for dehydration and overload -Laboratory studies including electrolytes, blood urea nitrogen, creatinine, and hematocrit -Replacement as necessary
nursing interventions for mucositis -Frequent, gentle oral hygiene, before and after meals -Soft toothbrush or if counts are low, sponge-tipped applicators -Rinse only with NS, NS and baking soda, or prescribed solutions -Perineal and rectal care
two most common side effects of chemotherapy nausea and vomiting
Many chemotherapy agents cause some degree of ____ (depression of bone marrow function), resulting in decreased WBCs (leukopenia), granulocytes (neutropenia), RBCs (anemia), and platelets (thrombocytopenia) and increased risk of infection and bleeding. myelosuppression
Decreased RBCs can lead to ____? What are treatments to correct this? anemia; folic acid, iron, transfusion
small waxy nodule with rolled, translucent, pearly borders; may appear shiny, flat, gray, yellow; most common type and recurrence is also common (rarely metastasizes); locally invasive malignancy arising from epidermal basal cells basal cell carcinoma
generally appears in sun exposed areas of the body (i.e. face, neck, hands, and scalp); localized tumor---therefore prognosis is good; least deadly---rarely causes morbidity or death basal cell carcinoma
rough, thickened, scaly tumor; may be asymptomatic or bleed; border is wide, more infiltrated, more inflammatory; arises from epidermis, sun damaged skin; commonly found on upper extremities/face/lower lip/ears/nose/forehead squamous cell carcinoma
prognosis depends on metastasis, type, level and depth of invasion; 4-8% metastasize by blood and lymphatic system; less aggressive than melanoma, can cause death squamous cell carcinoma
medical management: eradicate tumor, Tx depends on location/type/depth; alternatives: radiation, photodynamic therapy, topical chemotherapeutic creams; surgical: surgical incision, Mohs micrographic surgery, electrosurgery, cryosurgery basal cell carcinoma and squamous cell carcinoma
manifests as a change in nevus or a new growth on the skin; color is dark, red, blue colored or a mix; irregular shape; itching, rapid growth, ulceration, bleeding; cancerous neoplasm preset in dermis and epidermis melanoma
2x more common in men then women; found anywhere on the body (lower extremities – women; head, neck, trunk – men); Tx: surgical excision, chemotherapy melanoma
frequently r/t sun exposure; sun damage is cumulative and can manifest after a 20-50 year latency period skin cancer
blood flow to region or organ may be reduced; reduced blood flow leads to ischemia or infarction; ischemia/infarction may cause pain, swelling, and fever; crises are intermittent-aggravated by cold causing vasoconstriction and increased blood viscosity sickle cell anemia
entrapment of erythrocytes/leukocytes in microcirulation->tissue hypoxia, inflammation, necrosis d/t inadequate blood flow->perfusion resumes, substances are released causing oxidative damage to vessel->damage to the epithelium and vasculopathy develops acute vaso-occlussive crisis
from infection with the human parvovirus, hemoglobin drops rapidly and the marrow cannot compensate, leading to an absence of reticulocytes aplastic crisis
results when organs pool the sickled cells; spleen is often effected in children (splenic infarction happens often by 10 years of age, leaving spleen non-functional); liver and lungs are effected in the adult population sequestration crisis
IBD diet -low-residue, low-fat, high-protein, high-calorie diet -supplemental vitamin therapy and iron replacement -avoid cold foods -possible parenteral nutrition
dietary management for cholelithiasis -NPO, NG suctioning then soft, low-fat, high-carb diet (potatoes, bread, plain pasta) -avoid: eggs, cream, pork, fried foods, cheese, rich dressings, gas-forming veggies, alcohol
pancreatitis diet -enteral or parenteral nutrition may be needed -as acute Sx subside, oral feedings gradually reintroduced---high carb, low protein, low fat -b/w acute attacks--low-fat, high-protein -avoid heavy meals, alcohol, and excessive coffee and spicy foods
dietary management of peptic ulcer disease -avoid: extremes of food/drink temp, ETOH, coffee (including decaf), caffeinated beverages -eat three regular meals/day---small/frequent meals not necessary as long as antacid or H2 blocker is taken
post prostatectomy diet -encourage fluids -avoid coffee, alcohol, spicy foods
Created by: nurse savage
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