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Stack #38867

RRC Maintenance - COPD

QuestionAnswer
Emphysema hyperinflation of alveoli, destruction of alveolar walls, destruction alveolar capillary walls, narrowed/torturous small airways, loss of lung elasticity
Chronic bronchitis hyperplasia of mucous glands, hypersecretion of mucus, loss of cilia, chronic inflammatory changes and narrowing of small airways, altered function of macrophages
hyperplasia + hypersecretion of mucus + inflammatory response = narrowing of airway lumen and diminished airflow resulting in increased WOB
hypoxemia deficient O2 in blood
hypercapnia excessive CO2 in blood
Why does hypoxemia and hypercapnia develop in those with COPD? contricted bronchiole acts as physical barrier
What happens when there is an exacerbation of COPD inflammation!
Clinical manifestation of emphysema progressively worse dyspnea, minimal coughing, barrel chest, use of accessory and intercostal muscles to breathe, characteristically underweight, decreased breath sounds, limited diaphragmatic excursion
clinical manifestions of chronic bronchitis frequent productive cough/frequent clearing of throat, SOB, wheezing on forced expiration, frequent respiratory infections, normal weight or heavy-set
diagnostic tests for COPD Chest x-ray, pulmonary function tests, arterial blood gases, sputum analysis, CBC
Risk factors for COPD exposure to cigarette smoke, occupational exposure to dust, asbestos, silica, coal, gas fumes; chronic marijuana, pipe and cigar smoke, age
What s+sx of heart failure need to be taught to client increased dyspnea, fatigue, increased coughing, changes in amount and consistency of sputum, peripheral edema, fever or sudden weight gain
bronchodilators opens airway by relaxing smooth muscle of airways - resulting in increased airflow which may help loosen mucus
inhaled steroids to decrease inflammation and thereby increase airflow -- should only be used short-term to relieve symptoms during exacerbations
antibiotics given based on C&S results of sputum sent to treat underlying pneumonia
diuretics to reduce fluid overload in presence of cardiac complications: cor pulmonale
Give rationale for elevating HOB in semi-Fowlers during exacerbation maximizes ventilation and prolongs expiratory phase; also facilitates coughing and prevents aspirations
Give rationale for hydration during exacerbation of COPD liquifies secretions for easier expectoration
Give rationale for administering O2 and why we only give 1-2L/min via NP treat hypoxia --> use cautiously as those with chronic CO2 retention as hypoxemia stimulates respiratory drive, not hypercapnia
Give rationale for teaching pursed-lip breathing to a client with COPD may prevent airway collapse during expiration
Created by: bella83
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