Save
Busy. Please wait.
Log in with Clever
or

show password
Forgot Password?

Don't have an account?  Sign up 
Sign up using Clever
or

Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account.
Your email address is only used to allow you to reset your password. See our Privacy Policy and Terms of Service.


Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.
focusNode
Didn't know it?
click below
 
Knew it?
click below
Don't Know
Remaining cards (0)
Know
0:00
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

MedSurg #3

Honeynut718

QuestionAnswer
LIVER
Cirrhosis (slow onset) -is extensive, irriversible scarring/ the tissue becomes nodular and the nodes can block the bile ducts and the normal blood flow throughout the liver/ in early disease the liver is usually large,firm and hard/ as disease progresses the liver shrinks
Laennec's Cirrhosis - is caused by alcoholism
Postnecrotic Cirrhosis - is caused by viral hepititis and certain drugs or chemicals
Biliary Cirrhosis (Cholestatic) - is caused by chronic biliary obstruction from gallbladder disease.
Cardiac Cirrhosis - is caused by heart failure (rare)
Complications of Cirrhosis - depend on the amount of damage.
Compesated Cirrhosis - the liver is scarred but still can function without causing major symptoms
Decompensated Cirrhosis - liver function is impaired with other manifestations of liver failure
Common Complications of Hepatic Cell Damage (8) - portal hypertension/ ascites/ bleeding esophageal varices/ coagulation deficits/ jaundice/ portal system encephalapothy with hepatic coma/ hepatic-renal syndrome/ spontaneous-bacterial peritonitis
Portal Hypertension - increase in pressure within the portal vein develops as a result of obstruction to flow/ bloodflow backup can cause splenomegaly/ veins in the esophagus, stomach, intestines, abdomen and rectum become dilated
Ascites - free fluid containig almost pure plasma accumlates within the peritoneal cavity
Bleeding Esophageal Varices - fragile thin walled distended esophageal veins becomes irritated and ruptured/ most common in lower esophagus
Coagulation Defects - decrease synthesis of bile fat in the liver and prevents the absorption of fat solible vitamins, A D E & K, specially vitamin K because of clotting factors/ suspectible to bleeding and easy bruising
Jaundice - caused by one of two mechanism, hapatocellular jaundice( liver is unable to excrete bilirubin), intraheptic obstruction( edema, fibosis or scarring of the heptic bile duct channel interferes with bile and bilirubin excretion)/ jaundice is yellowing
Portal System Encephalopathy (PSE)or (Hepathic Coma) - end stage hepatic failure and cirrhosis is manifested by neurologic symptoms and characterized by altered LOC, impaired thinking and neruomuscular disturbance/ caused by elevated ammonia levels, treat with lactulose and nonabsorbable antibiotics
Hepato-Renal Syndrome - progressive oliguric renal failure associated with heptic failure, results in functional impairment of the kidnys with normal anatomic and morphologic features/ sudden ^in urine flow, ^serum urea nitrogen and creatinine levels and ^urine osmalarity,
Spontaneous Bacterial Peritonitis - manfestions include fever, chills, abdominal pain and tenderness
Splenomegaly - enlarged spleen results from backup of blood in spleen
Causes of Liver Disease - alcohol, viral hepititis, autoimmune hepatitis, steotohepititis, drugs and toxins, biliary disease, cardio disease, metabolic/gentic causes
Clinical Manifestations of Liver Disease - (early signs): fatigue, weight change, GI symptoms, abdominal pain, liver tenderness, pruitus (late):jauntice, icterus (orange eyes), dry skin, rashes, petechiae, warm bright red palms, spider angiomas, perpheral edema of the extremities and sacrum
Abdominal Assessment of Liver Disease - massive ascites, umbilicus protrusion, caput medusae( dilated abdominal veins), hepatomegaly (liver enlargement)
Hepatogemaly - can be assessed by palpating the RUQ, below the coastal border(ribcage)
Laboratory Assessment - asparte aminotransferase (AST) elevated, alanine aminotransferase (ALT) is more specific elevated and lactate dehydronase 140-280(LDH) elevated
Created by: Honeynut718
Popular Nursing sets

 

 



Voices

Use these flashcards to help memorize information. Look at the large card and try to recall what is on the other side. Then click the card to flip it. If you knew the answer, click the green Know box. Otherwise, click the red Don't know box.

When you've placed seven or more cards in the Don't know box, click "retry" to try those cards again.

If you've accidentally put the card in the wrong box, just click on the card to take it out of the box.

You can also use your keyboard to move the cards as follows:

If you are logged in to your account, this website will remember which cards you know and don't know so that they are in the same box the next time you log in.

When you need a break, try one of the other activities listed below the flashcards like Matching, Snowman, or Hungry Bug. Although it may feel like you're playing a game, your brain is still making more connections with the information to help you out.

To see how well you know the information, try the Quiz or Test activity.

Pass complete!
"Know" box contains:
Time elapsed:
Retries:
restart all cards