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N316 Fluids electrol
n316 exam 2Fluids and Electrolytes
Question | Answer |
---|---|
*Dehydration | "the excessive loss of water from the body." |
*hypercalemia | An abnormally high concentration of calcium in the blood. |
*fluid volume deficit | When fluid loss exceeds intake, a fluid volume deficit exists |
*older adult considerations | increase risk for FVD |
*Acidosis/alkalosis-application | (blank) |
Acidosis- | An abnormal increase in the acidity of the body's fluids, caused either by accumulation of acids or by depletion of bicarbonates. |
alkalosis | Abnormally high alkalinity of the blood and body fluids. |
*Metabolic/resp. | (blank) |
homeostasis | The body's tendency to maintain a state of physiologic balance in the presence of constantly changing conditions. |
Body fluid distribution H20 = [] % of body weight ICF = ? % and ECF = []?% | Body fluid distribution H20 = 60 % of body weight ICF = 40 % and ECF = 20% |
Body fluid distribution H20 = | 60 % of body weight |
ICF = ?? | E-lytes and 40% of body weight |
ECF = ?? | e lytes and 20% of body weight |
what E-lytes are found in ICF? | Potassium K+ magnesium Mg+ |
what E-lytes are found in ECF? | e-lytes Sodium Na+ Chloride Cl- calcium Ca+ |
ECF is classified by ? | Location |
Name the types of Ecf | interstital, intravascular, trancvellular |
define the following EFC fluid interstital, | fluid between cells |
define the following EFC fluid , intravascular, | fluid w/in blood vessels plasma |
define the following EFC fluid trancellular | urine, GI fluid, cerebral spinal fluid, pleural, synovial, intraocular etc. |
Normal fluid I & O Normal adult Intake = | 2500 mL/24 hrs. |
output = | 2500ml/ 24/ hrs. |
oral fluid intake = | 1200 cc/ 24 hrs. |
urine output = | 1500/24 hrs. |
water in food intake = | 1000/24 hrs. |
output respiration = | 500/24hrs. |
intake oxidation of food = | 300cc/24hyrs. |
output perspiration | 300/24hrs. |
cations= | + carged |
anions | - carge |
catinas, consist of | sodium,potassium,calcium,and magesium |
anions, consist of negetavely charged | psosphorous bicarbonate chloride |
mechanisms of body fluid movement | osometic preassure, hydrostatic preassure, diffusion, filtration, active transport |
osometic preassure, | power of a solution to draw h20 across a membrane high pressure gradient to low preassure gradient |
hydrostatic preassure, | |
diffusion, | The transport of matter from one point to another by random molecular motions. It occurs in gases, liquids, and solids. |
filtration, | The separation of solid particles from a fluidsolids suspension of which they are a part by passage of most of the fluid through a septum or membrane that retains most of the solids on or within itself. |
active transport | The passage of ions or molecules across a cell membrane against an electrochemical or concentration gradient, or against the normal direction of diffusion. |
body maintains homeostasis ? | thrist, kidneys, renin angiotensin/ aldosterone system, adh, ANF |
thrist, | felt when serum osmolarity > 295 |
kidneys, | volume and electrolyte balance osmalarity |
renin angiotensin/ aldosterone system, | intravascular fluid balance and blood preassure |
adh, | antidiuretic hormone regulates h20 excreation from the kidney |
ANF | atrial natriuretic factor releases when fluid is ovweloaded /to high |
roy behavior assessment | (oxygenation,nutrition, elimination, activity rest, protection, neurological alterations, labs |
roy stimuli assessment | chronic Illness, Medical intervention, cognator effectness, developmental-older adults*, enviromental |
why r older adults have an increased risk for FVD? | decrease perception thrist, - in body fluid amount, changes in body structure and function ie renal , temp regulation, incontinence, physical conditions/dissabalities, cognitive impaorments |
dehydration | loss of h20 alone |
hypovolemia | decrease in circulating blood volume |
Third spacing | a shift of fluid from vascular space into an unuseable space. |
causes of FVD | inadaquate fluid intake, failure of regulatory mechanism fluid loss, |
signs and symptoms of fvd | weight loss, thrist, postural hypotension, tachycardia, increase body temp, decrease pulse volume |
tachycardia | A rapid heart rate, especially one above 100 beats per minute in an adult. |
FVD diagnostic tests | concentrated urin SG> 1.030, decreased urine output, E-lytes, osmolarity, CVP sub normal, increased hemocrit elevated bun(possible |
nursing diagnosis in hypovolemia | Fluid volume deficit [r/t} inabality to maintain oral intake of fluids AEB vomiting [2] r/t lack of cognative abality to understand neeed to drink fluids AEB: confusion disorentation [3] r/t lack of info 2 replace fluids-Ineffective tissue perfusion, risk |
treatment hypovolemia | prevention, treat cause of deficits evaluate effectness of treatment |
treatment hypovolemia prevention at risk | elderly, children, persons with fluid loss (V/D) atheletes |
treatment hypovolemia treat cause of deficits | replace oral, iv, enteral, isotonic may need to add e-lytes |
fFE terminology hypervolemia, edema, causes | system failure,excessive intake of sodium, IV solution w/ NaCI |
fFE terminology hypervolemia, edema, system failure causes | heart, kidney cirrhosis of Liver, adrenal gland dissorders, corticossteroides, stress conditions causing a release of ADH/aldosterone |
ADH/aldosterone | antidiuateric hormone A steroid hormone secreted by the adrenal cortex that regulates the salt and water balance in the body. |
adrenal gland dissorders | water and electrolyte loss associated with this condition results from deficiency of the adrenal hormone, aldosterone |
corticosteroid | Any of the steroid hormones produced by the adrenal cortex or their synthetic equivalents, such as cortisol and aldosterone. Some corticosteroids regulate fluid balance in the body |
S/S of fluid excess | weight gain, circulatory overload peripheral edema, diagnostics |
S/S of fluid excess weight gain, | >5% over short period |
Diagnostics S/S of fluid excess chest x-ray | pulmonary edema |
Diagnostics S/S of fluid excess Serum Na and Osmolarity: | WNL Within Normal Limits |
Diagnostics S/S of fluid excess Hgb. and Hct | slightly below normal limits |
Diagnostics S/S of fluid excess may develop metabolic acidosis | if fails to adapt |
Diagnostics S/S of fluid excess low BUN | (blank) |
pulmonary edema | An effusion of fluid into the alveoli and interstitial spaces of the lungs. Edema of the lungs usually due to mitral stenosis or left ventricular failure |
Serum Na and Osmolarity | Serum = Watery fluid from animal tissue, such as that found in edema. Na = The symbol for the element sodium.Osmolarity = The osmotic concentration of a solution expressed as osmoles of solute per liter of solution. |
WNL | Within Normal Limits |
Hgb. and Hct | HGB (Hemoglobin) HCT (Hematocrit) |
BUN | blood urea nitrogen |
treatment of FVE | prevention, manage fluid intake, diuretics |
treatment of FVE diuretics | loop lasix,thiazides, osmotic diuretic, potassium sparing |
treatment of FVE loop lasix | inhibit Na reabsorption in ascending loop of henle |
treatment of FVE thiazides | diuril same as loop lasix but @ distal tubule less potent than loop same SE |
treatment of FVE osmotic diuretic | Mannitol (IV) |
treatment of FVE potassium sparing | spironolactone |
spironolactone | spi·ro·no·lac·tone (spī'rə-nō-lăk'tōn, spī-rō'-, spī-rŏn'ə-) |
adaptation to hypervolemia | left sided heart failure,right sided heart failure |
adaptation to hypervolemia left sided heart failure will present as | pulmonary edema crackles |
adaptation to hypervolemia right sided heart failure will present as | pweipheral edema pedal edema |
nursing diagnosis hypervolemia | (blank) |
electrolyte imbalances characteristics | mainly in ECF, normal values 135-145 mEq/L, |
ECF | extracellular fluid |
electrolyte normal lab values | 135-145 mEq/L |
electrolytes actions | regulates fluid volume, osmolality, maintains neuromuscular activity |
sources of electrolytes | (blank) |
electrolyte adaptation to imbalances | kidney saves or excretes stimulates renin and aldosterone system , ADH, glomercular filtration rate, natriuretic peptide release |
electrolyte adaptation kidney saves or excretes stimulates renin and aldosterone system why? | (blank) |
electrolyte adaptation kidney saves or excretes ADH, why? | (blank) |
electrolyte adaptation kidney saves or excretes glomercular filtration rate, natriuretic peptide release Why? | (blank) |
@next hyponatrumia | (blank) |
hyponatrumia | A serum sodium level of less than 136 mEq/L. A deficiency of sodium in the blood |
hypernatremia | >145mEq/L An abnormally high plasma concentration of sodium ions. |
*hyperkalemia | >5 mEq/L An abnormally high concentration of potassium ions in the blood. |
*hyperkalemia cause less common more dangerous | cardiac arrest, renal failure, medications, extensive tissue trtauma-burns, crush injuries, severe infections, rapid IV infusion |
pseudo hyperkalemia | (blank) |
S/S hyperkalemia | abnormal heart rate/rythem/ecg changes skeletal muscle weaqkness tremors, irritability GI diarrhea colic, Nero paresthesias flacid paralysis Collaborative care |
paresthesias | A skin sensation, such as burning, prickling, itching, or tingling, with no apparent physical cause. |
Calcium | 8.5 to 10mEq/L 99% bound to phosphorus to form minerals in bones and teeth only 1% extracellular and ionized (free) active |
actions of ionized Calcium | regulates muscle contraction and relaxation Maintains cardiac function, acts in blood clotting process |
Potassium | <3.5 mEq/L intracellular cation (K+) vital to cellular metabolism especially skeletal and cardiac muscle activity Daily intake needed, kidneys primary regulator aldosterone shifts in and out of cells in response to ph of the blood |
hypocalemia | <8.5mEq/L low calcium |
hypercalemia | serum calcium >10mEq/L |
hypercalemia causes | hyperparathyroidism malignancies lack of weight bearing w/ prolonged immobility self limited in a successful kidney transplant and excessive intake of ViD or Ca thiazide diuretics and renal failure |
thiazide diuretics | Any of a group of drugs that block reabsorption of sodium in the distal tubules of the kidneys, used as diuretics primarily in the treatment of hypertension. |
S/S hypercalemia | muscle weakness slow GI Abn heart rythm |
hypercalemia may lead to | peptic ulcer, kidneystones, cardiac arrest |
magnesium | 1.6-2.6mEq/L |
magnesium characteristics | mainly intracellular in bone, green veggies, excreated kidneys, vital 2 cellular function, affected by K and Ca levels |
K and Ca | K Abbrev. for potassium.Abbrev. Ca for calcium. |
hypomagnesia | <1.6mEq/L |
Acidosis | The condition where the hydrogen ion concentration increases above normal (reflected in a pH below 7.35). |
Alkalosis | The condition where the hydrogen ion concentration decreases below normal (reflected in a pH above 7.45). |
Alkalosis Acidosis application | (blank) |
acid base balancenormal p.h. | 7.35-7.45 |
body constantly produces acids | carbonic acid |
eliminated through lungs | as co2 |
lactic hydrochloric sulfuric | kidney |
most acids and bases are | weak |
the major base = | bicarbonate |
body constantly responds to regulate ph by | buffer systems, respiratory systems, renal (metaBOLIC) SYSTEMS |
buffer systems, | IMMEDIATE RESPONSE |
RESPIRATORY SYSTEMS | RESPONDS W/IN MINUTES |
RENAL (METABOLIC) SYSTEMS | RESPONDS HRS TO DAYS |
ARTERIAL BLOOD GAS MEASUREMENT PH NORMAL= () ACIDIC =() ALKALINE =() | PH NORMAL= =7.35-7.45 ACIDIC <7.35 ALKALINE = >7.45 |
Pco2 NORMAL = ACIDIC = ALKALINE = | NORMAL = 34-45 MMHg ACIDIC =>45 MM hG ALKALINE =< 35 mm Hg |
Pco2 | Partial Pressure of Carbon Dioxide |
HCo3 NORMAL = ACIDIC = ALKALINE = | NORMAL =22-26 mEq/L ACIDIC = <22mEq/L ALKALINE =>26 mEq/L |
HCo3 | Bicarbonate |
pO2 normal hypoxia | normal = 80-100mm Hg hypoxia<80 mm Hg |
pO2 | Partial Pressure of Oxygen |
hypoxia | Insufficient levels of oxygen in blood or tissue |
Acidosis | A state characterized by actual or relative decrease of alkali in body fluids in relation to the acid content; depending on the degree of compensation for the acidosis, the pH of body fluids may be normal or decreased; an accumulation of acid metabolites |
Acidosis | H ions concentration increases and pH <7.35 |
Acidosis respiratory | unable to get rid of CO2 |
Acidosis metabolic | excess acid or lack of bicarb |
alkalosis | A pathophysiological disorder characterized by H-ion loss or base excess in body fluids (metabolic alkalosis), or caused by CO2 loss due to hyperventilation (respiratory alkalosis). |
alkalosis | H ion concentration decreases below normal and ph > 7.45 |
alkalosis respiratory | exce4ssive co2 loss |
alkalosis | bicarb excess or loss of H ions |
symptoms of acidosis respiratory | ph <7.35, pCO2 > 45mm Hg resp hypoventilation is cause (likely Resp. can't respond) Neuro: HA, blurred vision, irritable, confused |
symptoms of Acidosis Metabolic : pH | Metabolic : pH < 7.35, HCO3 < 22mEq/L resp. hyperventaltion to blow off CO2, NEURO, HA, weak, fatigue, confusion, stupor, coma GI: N/V Skin:warm/ flushed CV Dysrhythmias d/t hyperkalema |
symptoms of alkalosis | respiratory ph>7.45, pCO2 <35mm Hg resp rapid shallow breathing cause NEURO panic light headed paresthesias of extremities lead to seizure LOC CV palpitions, chest tightness |
symptoms of alkalosis metabolic | ph>7.45, HCO3 >26mEq/L RESP response dec rate and depth of respirations NEURO; altered mental status numbness tingling at mouth & extremities muscle spasms may lead to seizure LOC CV: Arrhythmias d/t hypokalemia |