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DSCC Heart Failure

Heart Failure

QuestionAnswer
Causes of Left-sided HF HTN, CAD, valvular dz (mitral/aortic)
Causes of Right-sided HF Left ventricular failure, right-sided MI, pulmonary HTN
What does the sympathetic nervous system do to compensate for HF? Increases catecholamines (->hypoxia), beta adrenergic (increases HR), and alpha adrenergic (increases BP)
What is activated by decreased blood flow to the kidneys d/t HF? RAS - Renin-Angiotensin System
What is released by the RAS do in response to HF? Angiotensin II (causing vasoconstriction) and aldosteron (causing Na and H2O retention)
What are the bodies chemical responses to HF? MI:immune response releases tumor necrosis factor (TNF) and interleukins (IL-1 & IL-6); CHF: natriuretic peptides (B-type [BNP]) is released with fluid overload
Why does the heart enlarge (myocardial hypertrophy) in response to HF? To provide more muscle mass for more forceful contractions
S/S of left-sided HF: -Early manifestations: fatigue, activity intolerance -Dizziness, syncope -Dyspnea, SOB, DOE, cough, orthopnea, cyanosis -Oliguria (day), nocturia -Inspiratory crackles & wheezes -Pulmonary congestion & decreased cardiac OP -S3/S4 gallop
S/S of right-sided HF: -Weight gain, swollen hands/fingers -Increased BP -Lower extremity edema -Liver engorgement, RUQ pain -JVD -Impairment of right ventricle
S/S of pulmonary edema: -Disorientation/acute confusion -Anxiety/restlessness -Dyspnea at rest -Hyper- or hypo- tension -Tachycardia -Crackles -Dysrhythmias: PVCs (premature ventricular contraction) -COUGH WITH PINK, FROTHY SPUTUM
How to position pt with pulmonary edema: -Hi-Fowler's c legs down if not hypotensive
Nursing care for pulmonary edema: -Hi-flow O2 @ 5-6L/min by face mask or @ 10-15L/min by non-rebreather -Pulse ox & cardiac monitoring -May need CPAP or mechanical ventilation -NTG, SL q5 min x 3 doses if SBP > 100 -Obtain IV access -VS q30 min-1 hr
Meds given in case of pulmonary edema: -Lasix or Bumex -Morphine (decreased venous return, anxiety and workload of breathing
What is the best tool for dx HF? Echocardiogram
What monitors heart pressures? Pulmonary artery catheter
Nursing interventions for Ace inhibitors: Monitor BP, K levels (increased), and WBC (neutropenia)
Ace inhibitors end in: aPRIL
Which type of med for HF is given to decrease after-load, improve cardiac OP & renal blood flow? Ace inhibitors
Which type of med for HF is given to decrease pulmonary congestion & peripheral edema? Ace inhibitors
ARBS = Angiotensin II Receptor Blockers
Nursing interventions for ARBS: Monitor BP & K levels
The generic names of these meds end in -artan: ARBS
How do ARBS meds work? -Block the action of angiotensin II @ receptor -->decreased arterial resistance & arterial dilation -Block aldosterone -->preventing Na & H2O retention
Diuretics given c HF: Lasix, Aldactone, HCTZ
This drug improves contractility in HF: Digoxin (Lanoxin)
When would you hold a dose of digoxin? If apical HR is < 60
With which med would you assess apical HR for 1 full minute prior to admin? Digoxin
S/S of digitalis toxicity: -Anorexia -Fatigue -Bradycardia -Dysrhythmias -YELLOW HALOS
What would you administer if your pt showed s/s of digitalis toxicity? Digibind
These pts should be monitored especially carefully when on digoxin? Pts c renal failure
Dopamine and dobutamine are: Sympathomimetic agents
Why is dobutamine preferred over dopamine? It doesn't increase HR
Nursing interventions when giving sympathomimetics: Monitor BP
These meds are given by IV and can be titrated: Dopamin & dobutamine
These phosphodiesterase inhibitors end in: -cor
Which type of meds increase contractility and cause vasodilation (increasing cardiac OP & decreasing after-load? Phosphodiesterase inhibitors
You would not want to d/c these meds abruptly: Inocor & Primacor
Hepatoxicity & thrombocytopenia are risks for pts taking what med? Inocor
Impaired Gas Exchange r/t Heart Failure nursing interventions: -Monitor resp. status:rate, rhythm, quality -Auscultate breath sounds -Monitor O2 sats; provide supplemental O2 PRN -TCDB q2 hr & PRN -Maintain Hi-Fowler's positioning
Decreased Cardiac OP r/t HF nursing interventions for positioning: Elevate HOB
S/S of decreased cardiac OP and tissue perfusion: -Changes in LOC -Decreased urine OP -Cool.clammy skin -Diminished pulses -Dysrhythmias
Other nursing interventions for decreased cardiac OP r/t HF: -Monitor VS and O2 sats PRN -Monitor BNP levels; report trends -Auscultate heart & lung sounds -Admin O2 PRN -Admin meds as scheduled -Encourage rest, explain rationale -Avoid valsalva maneuver
Activity Intolerance r/t HF nursing interventions: -Organize nursing care to allow rest periods -Assist c ADLs PRN; encourage independence within prescribed limitations -Use passive & active ROM exercises; consult PT -Provide written & verbal info about activity after discharge
No limits: ordinary physical activity does not cause undue tiredness or SOB Class I
Slight or mild limits: comfortable @ rest, but ordinary physical activity results in tiredness or SOB Class II
Marked or noticeable limits: comfortable @ rest, but less than ordinary physical activity causes tiredness & SOB Class III
Severe limits: unable to carry on any physical activity w/o discomfort; symptoms are also present @ rest Class IV
Nursing Dx r/t HF: -Impaired Gas Exchange -Decreased Cardiac OP -Activity Intolerance -FVE -Ineffective Tissue Perfusion -Anxiety -Ineffective Therapeutic Regimen Management
Nutrition management for HF: -Limit Na intake to 2-3g/day -Limit fluid intake to 2L/day
Surgical management for HF: -Ventricular-assistive devices (VADs) -Heart Transplantation
Which med should pts c HF avoid? NSAIDS
Pre-Load (rubber band) Volume coming into ventricles(end diastolic pressure)
When is pre-load increased? -Hypervolemia -Regurgitation of cardiac values
After-Load (balloon) Resistance that left ventricle must overcome to circulate blood
When is after-load increased? -HTN Vasoconstriction-->increased after-load & increased cardiac workload
Actions of ACE inhibitors: Decrease peripheral vascular resistance w/o increasing cardiac OP, HR or contractility
S/E of Ace inhibitors: -Dizziness -Orthostatic hypotension -GI Distress -HA -COUGH
Beta-Blockers end in: -olol
Actions of beta-blockers: Blocks beta receptors in the heart causing decreased HR, force of contraction & rate of A-V conduction
S/E of Beta-blockers: -Bradycardia -Lethargy -GI Disturbance -CHF -Decreased BP -DEPRESSION
Treatment goals for HF: -Improve cardiac fx -Remove accumulated fluid & Na -Decrease cardiac demands -Improve tissue oxygenation
Created by: shall5
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