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chpt 38 O2 Perfusion
aneurysms, PAD, venous insufficiency
Question | Answer |
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Following abdominal aneurysm repair, the nurse should | maintain adequate B/P to avoid dissection, hydration, diuretics, hourly urine I/O, pulses, color, temp, capillary refill time, NPO, do not remove NGT till flatuence, ANTBX for 4-6 weeks for infx, no heavy lifting |
Pre-Op for aortic aneurysm repair | NPO, bowel prep (laxative, enema), preoperative ANBX, TUBES: A-line, catheters, EKG monitoring, pain meds, ANBX b/f to prevent infx |
what is PAD | advanced marker for advanced systemic atheroscerosis, progressive narrowing & degeneration of neck, extremities, & abd |
4 most significant causes of PAD | #1 smoking,hyperlipidemia, HTN, DM |
risk factors for PAD | obesity, hypertriglyceridemia, family hx, sedentary, stress |
Atherosclerosis pathophysiology | migration & replication of smooth ms cells, deposition of CT,lymphocyte, macrophage infiltration, accumulation of lipids |
Artherosclerosis causes 2 main things | 1. narrowing of artery-->insufficient blood supply to organ 2. d/t use of compensatory artery enlargement, aneurysm results |
Aortic aneurysm risk factor and etiology | highly genetic, etiology is atherosclerosis, |
pseudoaneurysm | enlargement of the outer layer of the blood vessel d/t leakage of blood. Enlargement of the outer layer only |
Thoracic aneurysm | usually asymptomatic, deep chest pain to nterscapula, dysphagia(swallowing), distended neck veins, edema in bilateral upper extremities, tamponaded by surrounding structures, Grey Turner sign |
Abdominal aortic aneurysm | often asympomatic, deadly, pain in abd to the back, may embolize plaque causing "blue toe,"massive hemmorhage into abd, hypovolemic shock w/tachycardia, pale and clammy, do not palpate abdominal mass |
size of aortic aneurysm | small:<4 cm--> risk factor modification, decrease BP, CT Q6 months large:>5.5 cm--> conventional surgery |
Aortic dissection | a tear in the intimal lining, more common in thoracis d/t HTN, "tearing pain" anterior chest to intrascapula, abd, legs. initial tx w/antihypertensive meds/lower contractility, sugery |
PAD lower extremities s/s | absent pulse, intermittent claudicatio (w/exercise), rest pain (surgery), parathesia,ulcer pain/shooting (oversensitive), have pt check feet,atropphy,thin/shiny skin, hair loss, elevation pallor, dependent rubor, gangrene, bony area/toes/forefoot,cold |
venous ulcer s/s | varicose/spider, ted hose/ACE, no claudication/rest pain, pruritis, damp to dry, skin masceration, elevate leg, pulses present, brown pigmentation, granulation present(pink). ankle area, superficial/uneven edges |
Intermittent claudication | classic symptom PAD d/t exercise, recurrent, resolves in 10 min or less, pt should walk till pain, rest & walk again |
PAD lower extremity dx: segmental blood pressure | B/P @ thigh below knee & ankle--> falloff >30mmHG+ PAD |
Ankle brachial index for PAD | ankle/brachial SBP Normal is 0.9-1.3 |
Risk factor Modification PAD | complete smoking cessation, tx hyperlipidemia (cholesterol <200- statin drugs), tx HTN & DM, HbA1C <7% |
Drug tx for PAD | antiplatelet- (ASA & Ticlid), ACEI(ramipril to increase walking), ASA & Plavix can be taken together |
2 drugs to tx intermittent claudication (PAD) | 1. Trental: dec blood viscosity 2. Pletal: inc vasodilation, inc walking |
most effective tx for intermittent claudication | walking 30-60 min daily and nutritional tx |
Ginko Biloga | increases walking distance for intermittent claudication |
folate, vitamin B6 and B12 | lowes homocystrine levels: >10micromoles/L associated w/atherosclerosis, strokes, MI, blood clots, & Alzheimers |
Critical limb ischemia | chronic w/ischemic rest pain, arterial leg ulcers, gangrene |
critical limb ischemia care | prevent trauma to leg, inspect/lubricate both feet, NO BATH/SOAKING d/t masceration avoid heat/cold,chemicals & heel pressure, no compression, cover w/dry dressing, dangle feet |
PAD lower extremities acute intervention | neuro check Q15 min then Q hour, loss of palpable pulses reported, KNEE FLEX AVOIDED, AMBULATION |
Acute arterial ischemia | sudden interruption to tissue/organ caused by EMBOLISM- tissue death if not tx immediately |
6 P's of Acute Arterial ischemia | pain pallor pulselessness parethesia poikilothermia(adaptation to external environment/cold) |
Acute ischemia tx | IV heparin- monitor PTT Q6H- does not dissolve clot- thrombolectomy balloon catheter used, surgical revasularization, amputation |
Buergers dz | thromboangitis obliterans, acute INFLAMMATION/thrombosis of hands/feet occlusive dz of median arteries ischemic,affects men, pain @ rest wake up at night must quit smoking or lose fingers/toes STRONGLY r/t smoking |
Buerger's dz medications | 1. vasodilators (minoxil, hydralazine), 2. CCB(procardia & Norvasc) 3. antiplatelet (ASA & Ticlid)4. Anticoagulation (Coumadin Trental)--> dec blood viscosity |
Raynaud's phenomenon | vasospasm of small arteries in fingers/toes, causes discoloration, young women age 14-40, triggered by stress, and cold, have pt wear warm, loose clothing, avoid caffeine/tobacco, CCBS (procardia & diltiazem) |
venous thrombosis classified as | DVT and thrombophlebitis |
thrombophlebitis | INFLAMMATION of vein (d/t IV) caused by IRRITATION not infection, swelling/red |
DVT occurs where... | occurs commonly in femoral and iliac veins |
Thrombosis etiology | virchow's triad: 1. venous stasis-dysfx valve/inactive extremity 2. endothelial damage (IV or trauma) 3. hypercoagubility- smoking/estrogen/steroids |
pathophysiology venous thrombus | RBC/WBC, platelets, & fibrin in valve cusp or vein, endothelial cells cover, thrombus may detach & become embolus |
how long for clot to be lysed by body | 5 days |
superficial thrombosis | palpable, cord-like, tender to touch,caused by trauma to varicose veins |
superficial thrombosis tx | NSAIDS, elevate extremity and apply moist heat |
DVT s/s | unilateral leg edema pain, erythmia, temp >100.4, PE is life threatening |
DVT tx | bed rest, prevention/prophylaxis (heparin/lovenox), elevation, warm compress, compression stocking |
coumadin & heparin used together b/c... | coumadin takes time to kick in, while heparin is immediate |
1. Coumadin 2. unfractionated heparin 3. fractionated heparin (LMWH) | 1. administer same time Q day, PO,check INR, vit k antidote 2. monitor PTT/ACT, given SQ or IV 3. no monitoring, given SQ, do not expel bubble w/lovenox, |
hemosiderin causes... | leg discoloration |
venous leg insufficiency | skin brown, thick, assess for PAD b/f tx w/compression stocking (NOT if PAD present too), moist dressing zinc, protein, vitamin c, skin graft, avoid standing, elevate leg above heart, post healing- daily walking program |
aortic dissection etiology | a false lumen is created between the intima and media. As the heart contracts, pressure increases the dissection, which could occlude blood flow to brain, abd, kidneys, spinal cord, and extremities. |
aortic dissection complications | cardiac tamponade--> when ascending aortic arch involved- blood goes to pericardial sac. Rupture could cause death. |
aortic dissection dx studies | similar to aortic abd aneurysm- MRI, CT, TEE, x-ray, Echocardiogram. After pt is stable--> angiography |
pt teaching for aortic dissection (meds) | lowering B/P and contractility, pain relief w/opioids (decrease anxiety, bed rest, IV beta blockers, control B/P w/nipride, CCB or ACEI, and to report s/e--> then surgical repair |
precautions w/anticoagulant therapy | IV unfractionated heparin given for acute arterial ischemic disorder. Pt should avoid taking NSAIDS/ASA, assess and report s/s bleeding. |