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Unit VII test
Paralytics/thrombolytics
Question | Answer |
---|---|
What is the formula for cardiac output? | Heart Rate * Stroke Volume Remember to conver to liters |
What are the two pharmacological agents for hypertensive emergencies? | Nitroprusside Sodium (Nitropress) Intravenous nitroglycerine |
Arteries | Cary oxygenated blood Resistant Vesssels Thick muscle |
Arterioles | Are small arteries |
Veins | Carry deoxygenated blood Less thick muscle Capacity vessles |
Veinules | Small Veins |
Capillaries | contain no smooth muscle Arterioles branch into capillaries at the tissues where gas exchange takes place. |
Thrombus | A clot the forms within a blood vessel or organ cavity such as the heart. |
Thromboembolism | A traveling thrombus in a vein |
Beta Blockers | Decrease myocardial oxygen consumption, cardiac output, heart rate, blood pressure and afterload. |
Acebutolol Beta Blocker | Secretal |
Atenolol Beta Blocker | Tenormin |
Metoprolol Beta Blocker | Lopressor |
Propranolol Beta Blocker | Inderal LA |
Anticoagulant | Prevents the formation of the fibrin clot by interfering with one of the steps leading to fibrin formation. |
Thrombolytics (Fibrinolytics) | Actually dissolve and liquefy the fibrin of the existing clots |
Anti-Coagulants | Heparin/ Warfarin (indirect) Lepirudin (Direct Acting) |
Alteplase Thrombolytic | Activase (tpa) |
Reteplase Thrombolytic | Retavase (tPA) |
Streptokinase thrombolytic | Streptase |
Tenectaplase thrombolytic | TNKase |
Depolarizing neuromuscular blocking agents | Bind to Ach receptor site initiate a massive depolarization of the muscle. Cause initial muscle fasciculations short acting |
Non-depolarizing neuromuscular blocking agents | Competitive antagonist binds to a receptor site Ach would occupy. Prevents motor end plate from depolarizing Non selective Can be reversed. Can also affect autonomic nervous system. |
Ultrashort duration | succinylcholine (Anectine) |
Intermediate Duration | Cisatracurium (Nimbex) Vercuronium (Norcuron) |
Long Duration | Pancuronium (Pavulon) |
What is the neurotransmitter for the somatic nervous system? | Ach |
What breaks down Ach? | Acetylcholinesterase (Ache) |
The somatic nervous system has receptors where? | The skeletal muscle |
What are three benefits associated with neuromuscular blocking agents? | 1. Reduce spontaneous breathing. 2. Remove movement that can dislodge tubes. 3. Reduce 02 consumption with patients with poor oxygenation. |
What are two more beneftis associated with neuromuscular blocking agents? | 1. Improve patient synchrony with the ventilator. 2. Used to facilitate intubation. |
What is the neuromuscular blocking agent that is depolarizing and can be reversed? | Succinylcholine (Anectine) |
The first nonpolarizing nueromuscular blocking agent. | Pancuronium (Pavulon |
The second nonpolarizing neuromuscular blocking agent. | Vecuronium (Norcuron) |
The third nonpolarizing neuromuscular blocking agent. | Cisatracurium (Nimbex) Broken down in the blood stream less effects on liver/kidney. |
Hypnotic | A medication that induces sleep |
Sedative | A medication that reduces CNS arousal |
Anxiolytic | Any medication that reduces the symptoms of anxiety |
Induction | relates to the time it takes to create the appropriate level of anesthesia |
Maintenance | Relates to continuation of the anesthetized state |
Termination | Refers to the time it takes for a patient to recover from anesthesia |
First stage of anesthesia | Analgesia- Conscious Sedation |
Second stage of anesthesia | Loss of consciousness (Reflexes like cough are present) |
Third Stage of anesthesia | Surgical anesthesia (complete respiratory depression) (loss of blinking reflex/eye movement) |
Fourth stage of anesthesia | Complete loss of respiratory drive, loss of cardiovascular tone. |
The antagonist of opoids | Naloxone (Narcan) |
Doxapram Ventilatory stimulant | Postanesthesia for drug-induced respiratory depression |
Ceffeine Ventilatory stimulant | Apnea of prematurity |
Common antidepressents | Prozac Paxil Celexa Zoloft Norpramin Pamelor Tofranil |
What are the four types of pain receptors? | Stretch Temperature Deep pressure CHemical |
Alprazolam Benzodiazepine | Xanax |
Chlordiazepoxide Benzodiazepine | Librium |
Diazepam Benzodiazapine | Valium |
Midazolam Benzodiazapine | Versed |
Lorazepam Benzodiazepine | Ativan |
Benzodiazepines | have sedative, hypnotic and anxiolytic affects |
Narcan | Naloxone |
Versed | Midazolam |
Diprovan | Propofol |
Valium | Diazepam |
Pavulon | Pancuronium bromide |
Nimbex | cistracurium besylate |
Norcuron | Vecuronium |
Propofol Anesthetic | Diprivan |
Ketamine Anesthetic | Ketaler |
Inhalation anesthetic | N20 |
First category to treat hypertension ACE Inhibitors | Causes vasodilation |
Second category to treat hypertension Diruectics | Decrease in blood volume |
Third category to treat hypertension B-Blockers | Decrease Cardiac Output |
Fourth category to treat hypertension Calcium-CHannel BLockers | Vasodilation |
Baroreceptors | pressure sensors |
Stretched to far | sense high pressure send signals to decrease sympathetic tone and reduce blood pressure |
Stretched too little | Sense low pressure Send signals to increase sympathetic tone and increase blood pressure |
What is the test to make dosing adjustments to heparin | Prothrombine Time (PT) |
What is the test to make dosing adjustments to warfarin | Partial prothrombine time (PTT) |
What are the four side effect of opoid analgesics | Respiratory depression (Apnea) Circulatory Depression Shock Respiratory arrest (Cardiac arrest) |
The reversal agents to non-depolarizing neuromuscular agents | Neostigmine Pyridostigmine |
Nociceptive nervous system | organ system responsible for dealing with pain |
Analgesia | Refers to the reduction in the sensation of pain |
Ventilatory stimulants | increase the rate and depth of respirations |