Heart/hemodynamics/blood
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each of the black spaces below before clicking
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P wave | show 🗑
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show | straight line from p-q
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show | ventricle depolarization
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show | ventricle repolarization
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P wave atrial contraction | show 🗑
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QRS ventricular contraction | show 🗑
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coronary arteries bring what to heart | show 🗑
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show | *left main coronary artery --> circumflex branch --> LAD left anterior descending
*right coronary artery
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if heart doesn't get enough 02, will switch to anaerobic metabolism which activates this rsn | show 🗑
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ischemia | show 🗑
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show | tissue death due to lack of 02 (blood supply)
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show | a tear in endothelium/plaque that then clots
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myocardial infarction | show 🗑
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show | decreased pumping ability
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Right sided heart failure s/s | show 🗑
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Left sided heart failure s/s | show 🗑
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Tx of heart failure, want to increase pumping ability or tx symptoms | show 🗑
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show | heart rate x stroke volume
*volume ejected per minue
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cardiac output is how many liters/minute | show 🗑
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show | 1-preload
2-contractility
3-afterload
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show | amount of blood entering ventricle during diastole (Frank Starling law)
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show | within limits, the greater the volume into heart during diastole, the greater the volume out during systole (not true during heart failure)
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what increases preload? | show 🗑
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What decreases preload? | show 🗑
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contractility defn | show 🗑
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what increases contractility? | show 🗑
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show | *decreased Ca
*barbiturates
*old age
*MI which leaves scar tissue
*heart failure
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if i increase contractility, what happens to stroke volume | show 🗑
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show | resistance the ventricle has to overcome in order to eject its blood
resistance ventricle has to overcome to eject its blood
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show | decreases, it has to push harder to eject same amount of blood
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show | decrease sv
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show | factors increasing after load
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show | *vasodilating drugs will decrease resistance thereby decreasing afterload (which will lower bp)
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factors effecting heart rate | show 🗑
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input to cardiovascular center from where? | show 🗑
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show | *cardiac accelerator by symp nerves
*cardiac decelerator by CN X vagus psymp
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show |
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show | *cardiac output
*peripheral resistance
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show | BP=CO x PR
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show | 1. lumen diameter
2. viscosity of blood
3. vessel length bwo # of capillaries which actually create resistance (as in obese people have more capillaries, why theres increase pv, inc pv --> bp
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show | CO x PR (cardiac output x periph resis)
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show | 1/3 systolic bp + 2/3 diastolic bp
*must be >60 for adequate brain/tissue perfusion
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show | pressure on artery walls during ventricular systole
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diastolic bp is | show 🗑
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show | how much blood is out there
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diastolic bp indicates | show 🗑
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show | *medulla oblongata is location of basomotor center of medulla oblongata bwo
glossopharyngeal and vagus nerves
*also RAA, ADH involved
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orthostatic decrease in bp temporarily decreases what? | show 🗑
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circulatory shock defn | show 🗑
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3 categories of circ shock | show 🗑
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show | 1. anaphylactic (histamine binds to vasodil)
2. neurogenic (withdraw of symp ns, most likely in spinal cord injury of thoracolumbar where symp nerves are)
3. Toxic shock causes vasodil
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cardiogenic shock is a | show 🗑
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Created by:
lorrelaws
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