Save
Busy. Please wait.
Log in with Clever
or
show password
Forgot Password?
Don't have an account?  Sign up 
Sign up using Clever
or
Username is available taken
show password


Make sure to remember your password. If you forget it there is no way for StudyStack to send you a reset link. You would need to create a new account. Your email address is only used to allow you to reset your password. See our Privacy Policy and Terms of Service.

Already a StudyStack user? Log In

Reset Password
Enter the associated with your account, and we'll email you a link to reset your password.
focusNode
Didn't know it?
click below
 
Knew it?
click below
Don't Know
Remaining cards (0)
Know
0:00
edit
Embed Code - If you would like this activity on your web page, copy the script below and paste it into your web page.

  Normal Size     Small Size show me how

Cardio defect embryo

QuestionAnswer
Membranous VSD Faulty formation of membranous VSD. L-->R shunt, with pulmonary HTN and excessive fatigue, exertion
Eisenmenger Occurs because of faulty VSD which leads to increased in pulmonary HTN. This leads to narrowing of pulmonary arteries. R--> L shunt develops with cyanosis
Muscular VSD Single/multiple perforations in the musclar IV septum
Common ventircle (cor triocularare biatrium) failure of the membranous/muscular IV to form
X-ray of VSD increased size of pulmonary artery and cardiomegaly
ASD findings S1 with fixed split S2, L--> R shunt
Foramen primum defect failure of the AV septum to fuse with the septum primum (abnormal mitral valve)
Tricuspid atresia AV defect (hypoplastic right heart). Insufficient amount of tissue to form tricuspid valve causing complete agenesis of the tricuspid valve . Cyanosis, patent foramen ovale, IV septal defect occurs
Ebstein's anomaly failure in the development of the tricuspid valve
Persistant truncus arteriosus AP septum doesn't completely form. Both ventricles pump into the aortic arch
D-Transposition of vessels Co-truncal septum fails to follow its normal spiral course and runs straigt down. Aorta orignates from R. Ventricle, pulmonary artery originates form L. ventricle. Ductus arteriosus remains open.
L-transposition of vessels Left and right ventricle are mixed up. Normal blood flow occurs
Tetrology of fallot Unequal divison of conus. Reuslts in 4 CV alterations: 1) narrow r. ventricle outflow tract (pulmonary infundibular stenosis), 2) hypertrophy of the R. ventricle wall because of higher pressure on R. Side, 3) IV defect, 4) overriding aorta (more to r)
Created by: ddecampo
Popular USMLE sets

 

 



Voices

Use these flashcards to help memorize information. Look at the large card and try to recall what is on the other side. Then click the card to flip it. If you knew the answer, click the green Know box. Otherwise, click the red Don't know box.

When you've placed seven or more cards in the Don't know box, click "retry" to try those cards again.

If you've accidentally put the card in the wrong box, just click on the card to take it out of the box.

You can also use your keyboard to move the cards as follows:

If you are logged in to your account, this website will remember which cards you know and don't know so that they are in the same box the next time you log in.

When you need a break, try one of the other activities listed below the flashcards like Matching, Snowman, or Hungry Bug. Although it may feel like you're playing a game, your brain is still making more connections with the information to help you out.

To see how well you know the information, try the Quiz or Test activity.

Pass complete!
"Know" box contains:
Time elapsed:
Retries:
restart all cards