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❤️ Meds and BP

Mind blown

QuestionAnswer
Normal < 120 < 80
Pre HTN 120-139 or 80-89
HTN1 140-159 or 90-99
HTN2 160 or higher or 100 or higher
HTN Crisis > 180 or > 110
Sequence of events 1systole + 1 diastolic = 1 heartbeat Cardiac cycle
The volume of blood pumped by 1 ventricle in 1 minute Cardiac output
Pressure of blood in the circulatory system measured for a diagnosis. Related to the force and rate of the heart beat and the diameter and elasticity of the atrial walls Blood pressure
Removes excess H2o and Na+ from your body, 2nd use for BP Diuretic medication
Hydrochlorothiazide (mainly used) HCTZ
Removes more sodium and H2O (Lasix) Furosemide
Drugs that block non-epinephrine and epinephrine ( adrenaline). Reduces HR BETA Blockers
Reason for using Beta Blockers To reduce heart rate and blood pressure
Beta blocker medication ends in LoL
Angiotensinogent + Renin = Angiotensin -> metabolizes -> Angiotensin 2
Angiotensin is a protein created and released by The liver
Angiotensin 1is a precursor for Angiotensin 2
As angiotensin is passed in the bloodstream through the lungs and kidneys. It's further metabolized by the action of angiotensin converting enzyme following binding to with your scepters found in most tissues of the body Angiotensin 2
Angiotensin 2 affects Construction of blood vessels, elevation of BP, stimulates the release of aldosterone from the adrenal cortex, kidneys, sodium + retention
ACE & ARBS are used for Kidney protection
Renin Angiotensin Aldosterone System(RAAS) Regulator of BP determinant of target organ damage, controls fluid and electrolyte balance through coordinated effects on the heart one vessels and kidneys.
Is the main factor of the RAAS and exerts its vasoconstrictor effects pre-dominantly on the postglomerular arterioles Angiotensin 2
Increases glomerular hydraulic pressure and ultrafiltration of plasma proteins, and fats that may contribute to the onset and progression of chronic renal damage Angiotensin 2
Angiotensin converting enzyme (ACE) inhibitors Medication that slow (inhibit) and have it parentheses the activity of the enzyme (ACE), which decreases the production of angiotensin 2
Type of medication also use the help diabetics with their strong medication. Acts to lower blood pressure ACE Angiotensin Conversion Enzyme
ACE medications end in (Pril) ex. Benazepril, Captopril, Enalapril.
ARBS Angiotensin 2 Receptors Blockers
Potent chemical formed in the blood that causes the muscles surrounding blood vessels to contract Angiotensin Receptors Blockers
ARBS medication ends in (Tan)
Relax blood vessels and increase the supply of blood and oxygen to the heart while reducing the hearts workload. Prevent calcium from entering the cells of the heart and blood vessels walls, & lowers blood pressure Calcium channel blocker's
Calcium channel blocker medication Ends in Pine ex. Amlodipine, Nicardipine, Diltiazem
Class 1A A-Fib/flutter, SVT, V-tach
Class 1A medications Quinidine, Procainamide, Disopryamide
Class 1B V-tach
Class1B medication Lidocaine, Tocainide, Meziletine.
Claas1C Life threat SVT, V-tach
Class1C medication Flecainide, Propafenone, Moricizine,
Works by directly acting on the cell membranes of the heart fibers Glycosides
Potent inhibitors of sodium and potassium which increases the polarization of the myocardial cells Digitalis glycosides
A medication mainly used for A-fib Digitalis Glycoseides
(Lasix) Furosemide is a medication for what symptom Edema
Black from binding to receptors on myocardial cells ARBS
Reduce his heart, lowers action potential, med's and in Zam and Pine, prevents calcium from entering cells the heart and Blood vessel walls resulting in lower price pressure Calcium channel blockers
Blocks the fast sodium channels, responsible for fast Depolarization of the Fast response of the action potential. Na+ Sodium channel Blockers
Created by: Georgia1984
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