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Platelets (Hema2)

Cards for Medical technology students currently taking Hema2 on Platelets

QuestionAnswer
Predisposition to thrombosis secondary to a congenital or acquired disorder thrombophilia
A clot that breaks free and begins to travel around the body is known as embolus
formation of a thrombus (blood clot) within a vein. Venous thrombosis
blockage of the main artery of the lung or one of its branches by a substance that has travelled from elsewhere in the body through the bloodstream Pulmonary embolism
most commonly results from deep vein thrombosis that breaks off and migrates to the lung venous thromboembolism
formation of a thrombus within an artery. In most cases, arterial thrombosis follows rupture of atheroma, and is therefore referred to as atherothrombosis.
causes a blood stasis within the atria with easy thrombus formation. atrial fibrillation
Factors that Affect Evaluation Anticoagulant therapy  Recent thrombotic events  Testing for AT, PS, PC, Factor VIII, and LA must be performed 10 to 14 days after therapy
Family of Ab that bind protein-phospholipid complexes  Include: Lupus anticoagulant (clot-based assay) ➢ Anticardiolipin (immunoassay) ➢ Anti-β2GPI (immunoassay
an immune response to nonself antigens from members of the same species, which are called alloantigens or isoantigens Alloimmune
happens when the body's natural defense system can't tell the difference between your own cells and foreign cells, causing the body to mistakenly attack normal cells Autoimmune
PTT with low reagent phospholipid Factor XII
DRVVT (Dilute Russell viper venom time) Factor X
KCT (Kaolin Clotting Time) Factor XII
DTT (Dilute Thromboplastin Time) Factor VII
 Mutation  Cause for increased thrombosis  Assay Prothrombin G20210A
risk factor for arterial thrombosis Plasma Homocysteine
Homozygosity for the MTHFR C677T mutation is associated with ______________but is not an independent thrombosis risk factor. Homocysteinemia
The plasma is first treated with a reducing agent to convert all disulfide forms to homocysteine, which becomes bound to fluorescein to form a detectable fluorescent derivative (not standardized) High-performance liquid chromatography
plasma disulfides are reduced, the homocysteine is converted to S-adenosylhomocysteine, and the S-adenosylhomocysteine is bound to an antibody conjugated to a fluorescein tracer. Enzyme immunoassay
TRUE OR FALSE. Plasma homocysteine rises by 10% per hour if blood is kept intact at room temperature after collection. TRUE
Plasma collection may be accomplished using heparin, sodium citrate, or ethylenediaminetetraacetic acid (EDTA) anticoagulants.
is a quantitative, clot-based, functional assay. The assay measures the ability of fibrinogen to form fibrin clot after being exposed to a high concentration of purified thrombin. Clot based method for Clauss
High fibrinogen concentration predicts hypercholesterolemia
Makes blood more viscous (favors coagulation, plt activation, and formation of atherothrombotic lesions) Fibrinogen activity
s a rare and serious condition that disrupts your blood flow. It is a blood clotting disorder that can turn into uncontrollable bleeding. Disseminated intravascular coagulation (DIC)
is the primary culprit of DIC because it activates platelets, activates coagulation proteins, and catalyzes fibrin formation, of which the ensuing clots consume control proteins. Circulating thrombin
In DIC, Thrombin cleaves fibrinogen, creating ________. Fibrin monomers
Free plasmin digests ... V, VIII, IX, and XI
is seen in meningococcemia, chickenpox, and spirochete infections. Purpura fulminans
____ and ________ immunoassays are sensitive and specific for thrombosis, which occurs in DIC, septicemia, eclampsia, pancreatitis, leukemia, liver disease, and trauma. Thrombin-Antithrombin (TAT)  Prothrombin fragment 1 + 2 (PF 1+2)
Adverse effect of treatment with unfractionated heparin eparin induced thrombocytopenia with thrombosis (HIT)
Created by: aphelion
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