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585-1

Stroke

TermDefinition
1. R CVA results in 2. L CVA results in 1. left hemiplegia 2. right hemiplegia
ischemic stroke 80% Includes ischemic necrosis and infarction May be due to hypo-perfusion Blockage of blood vessels in the brain
ischemic stroke physiology Atherosclerosis- in branches of vessels Thrombosis Embolism Cardiac- afib, arrythmia
occurrence of ischemic stroke Progressive symptoms Onset hours or days, often at sleep or rest Mild arm numbness/ morning paralysis Transient ischemic attacks present before
5 warning signs of stroke Vision issues Weakness Trouble speaking Headaches Dizzy
acute ischemic stroke management Stroke ambulance to rural areas Determine nature & size Administer TPA- breaks clot up, best within 1h, up to 6h post onset Surgery to remove clots Anticoagulation
prognosis of ischemic stroke Depends on extent/ location, with/ without coma In general will plateau after 5 or 6 mo up to 10y Good recovery rate for first few months
middle cerebral artery (MCA) Supplied by internal carotid, supplies lateral aspect of brain Frontal, parietal, temporal, occipital Most common stroke site Motor, sensory, cognition Possible homonymous hemianopsia
homonymous hemianopsia Visual field loss in same halves of both eyes
homunculus injury area 1. MCA sensory & motor 2. ACA sensory & motor 1. Arm, hand, face, tongue 2. Trunk, leg, foot, genital
MCA functional deficits Contralateral hemisensory loss Hemiplegia Visual field Apraxia, perseveration Poor judgement Apathy L CVA: Broca's or Wernicke's aphasia
Broca's vs. Wernicke's aphasia Limited expression vs. Limited reception/ comprehension
lacunar stroke Penetrating arteries have a blockage Only motor or sensory deficits Affects face, arm, leg
lenticulostriate arteries stroke Pure motor stroke Clumsy hand syndrome Ataxic hemiparesis (2nd most frequent type of lacunar stroke)
lateral thalamus/ parietal white matter stroke Pure sensory stroke Somatosensory loss to all primary modalities in face, arm, body If affects basal ganglia, then parkinson like symptoms may occur
anterior cerebral artery (ACA) Supplies medial aspect of brain Frontal and parietal areas Motor & sensory, legs, bowel, bladder, shoulder Behavioral disturbance- behavioral inhibition
ACA functional deficits Behavioral disturbance Apraxia Contralateral hemisensory loss Hemiparesis in foot Stopping of speech if left CVA
anastomoses Connection of distal ends among cerebral arteries Connection of ACA and MCA If you have blockage of MCA, ACA can supply
maximal ischemia AKA carotid border-zone syndrome Thinnest areas, most prone to blockage Corresponds to shoulder problem/ upper arm Boarder zone btwn MCA & ACA Motor recovery should be used
posterior cerebral artery Supplied by vertebral artery then basilar artery Temporal & occipital
PCA deficits Alexia- reading issues Anomia- word retrieval issue Visual agnosia Propagnosia Memory impairment Homonymous hemianopsia
wallenberg's syndrome Brainstem stroke Vertebral/ cerebellar artery Pain, temp loss, dry cold face on affected, ataxia, facial sensory loss No significant weakness
vertebrobasilar stroke Vertebral & basilar artery Posterior portions of brain affected
vertebrobasilar stroke occlusion leads to Visual disturbance Ataxia Clumsy Diff judging distance Memory loss Paralysis, local numb Impaired temp sensation Dizzy Diff swallowing
1. subarachnoid hemorrhage 2. subdural hematoma 3. epidural hematomas 1. cerebral arteries start bleeding, most popular 2. tearing of bridging veins btwn dura matter & arachnoid matter, due to trauma 3. torn meningeal artery, btwn dura matter & cranium, due to trauma
causes of bleeding Rupture Congenital factors Infection Tumor
arteriovenous malformations In any part of brain, brainstem, spinal cord Vessels grow into capillary bed, may burst as it becomes larger Age ~30 Present with seizure & headache Treated by surgery
ruptured intracranial aneurysms Small ballooning or dilations of vessel wall Ruptures in wall of artery Due to weak wall, high BP Often where there is branching No signs/ symptoms During waking hours Sudden severe headache Often age 40-65
hemorrhagic stroke mechanisms Once bleeding is controlled, clots occur then break down and removed If pressure is low, brain tissues will heal If severe bleed, may increase fatality More common in younger people Not predictable in terms of recovery
prognosis of hemorrhagic stroke Varied No coma + no LOC + no ventricle bleeding means good prognosis 1st episode of ICH- ~20% fatality Rebleeding increases fatality up to ~60%
rehab Capitalize on neurogenesis and synaptogenesis 3mo- years Motor learning Early mobilization once condition is stable Intensity & stages vary
acute phase rehab Early mobilization Ensure scapula glides freely Lower risk of other complications with transfers, appropriate bed & seating, skin management Maintain tissue length Return to ADLs Education Fall prevention
rehabilitation phase Postural control with maintain balance Encourage bilateral UE to help with balance Graded reaching activities Motor learning & cognitive rehab Use tech Driving & sexuality have specific programs
approaches 1. bottom up 2. process specific 3. top down 4. task specific 1. train component skills 2. assumes transfer of training will occur 3. emphasize intact skill training 4. client driven, repetitive task practice
environmental setup Tasks relevant to client needs as starting point Underlying deficits challenged via task Assistive devices for compensation
assessments COPM- goals ADL- FIM, barthel index Physical- MMT, AROM, PROM, gino, sensation, proprioception, dynamometer Cranial- not by OT Cognition Visual-perceptual Performance based observational ax
chedoke- mcmaster stroke ax Physical ax For people from 1wk to several yrs post stroke 7 stages of motor recovery to guide ax & tx
chedoke 2 components Impairment inventory- presence & severity of common physical impairments following CVA Disability inventory- measures functional outcome, gross motor function & walking
Created by: craftycats_
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