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Neuro3 Brainstem
Neuro3 Brainstem - Midbrain
Question | Answer |
---|---|
What structure is located just b/w the Thalamus and the midbrain? (medial to the medial Geniculate) | Pretectal Region. |
Swelling of what structure could compress the midbrain? | Pineal Gland. **Also I am sure cerebellar and Cerebral Swelling would also affect the midbrain. |
What is making up the bulk of the raustral pons? | 1.Corticospinal fibers (anteriorly). 2.Corticonuclear fibers. **1&2 are called transverse pontine fibers. |
What structure wraps around the Superior Cerebellar Peduncle? | The Medial Lemnicus, ALS, Lateral Lemniscus ribbon |
What two structures are located just inside the Superior Cerebellar Peduncle (in order from Anterior to Posterior) | 1.Central Tegmental Tract (CTT). 2.MLF (just anterior to the 4th ventricle) |
What type of fibers are contained within the Superior Cerebellar Peduncle? | Cerebellothalamic fibers. |
Where does the Superior Cerebellar Peduncle Decussate? | Midbrain |
Where does the Medial Geniculate body recieve fibers from? | Inferior Calliculus (auditory) |
What structure is located just anterior to the Cerebral Aquaduct in the midbrain? | Occulomotor Nuc. |
What is the Edinger-Westphal Nuc in control of? where is it located? | PNS control over the ciliary body/muscle and the pupillary sphincter muscle. **It is the medial most portion of the occulomotor nucleus. |
What is te black pigmented structure located just inside (posterior) to the Cerebral Peduncle? What does it produce? | Substantia Niagra. **Produces Dopamine. |
What structure is located just inside (posterior) to the Substantia Niagra? | Red Nucleus. |
What structure connects the two lobes of the thalamus posterior to the base of the 3rd ventricle? | Posterior commisure. |
Both III and IV enter the orbit via Superior Orbital Fissure, which does NOT pass through the Annulus? | IV (innervates the SO muscle). |
How will a patient present with Trochlear Palsy? | Head will be tipped towards the Contralateral (unaffected) side, elevating the affected eye and lowering the unaffected due to: 1.Ipsilateral eye is extorted OUT & UP. 2.Diplopia on Forward gaze |
List two Vascular compression Neuropathies and the structures involved | 1.Occulomotor: CNIII compression b/w Posterior Cerebral A and Superior Cerebellar A (could present intermittently due to BP). 2.Trigeminal Neuralgia: CN V is compressed by Superior Cerebellar A (very painful) |
How will Cavernous Sinus Syndrom from a Thrombosis present? | ACUTE ONSET: 1.Opthalmopalgia. 2.Complete drooping of the eye (CN III palsy). 3.Numb, painful patch of forehead 4.Ptosis. 5.Dilated pupil **Slow Onset: tumor, Aneurysm. Subacute: Infection |
What is the most common cause of Cavernous Sinus Syndrome? | Neoplasm (slow onset). |
How will the eye present with a complete CN III Palsy? | Down, out, and Dilated. (only have LR and SO). |
Decribe how the fiber radiations from both the Occulomotor Nuc and the edinger-westphal Nuc can cause different affects if damaged? | 1.Occ Nuc: Radiations run more laterally. Lesion here will cause occulomotor palsy w/ normal pupillary light reflex. 2.EW Nuc: fibers run more medially. Medial lesion would only damage PNS controll (pupil will be dilated w/ normal muscle control). |
Track the Pupillary Light Reflex | 1.Retina. 2.Optic N. 3.Optic Tract. 4.Lateral Geniculate. 5.Pretectal Region. 6.Edinger-Westphal's Nuc. 7.CN III. 8.Pupillary Sphincter muscle. |
What structure is damaged in Marcus-Gunn Pupil? When is it commonly seen? | OPTIC NERVE (Effects wont be seen if in the optic tract). **commonly seen with MS b/c of the high amt os myelin in the optic nerve. |
Describe the mechanism behind Marcus-Gunn Pupil? | 1.Direct light stimulation: Normal constriction. 2.Indirect light stimulation: Normal constriction. 3.Quickly back to direct light stimulation: Abnormal dilation. |
Describe the presentation of a person with a vascular lesion in the Paramedian territory | PARAMEDIAN SYNDROME: 1.Oculomotor palsy (III Nuc damage). 2.Bilateral limb ataxia (cerebellarthalamic fiber damage). 3.Triad (Thalamus damaged): Memory loss, Dementia, Sleep disturbance. **Usually see 1&2 or just 3. |
Describe the presentation of a person with a vascular lesion in the Short Circumferential territory | WEBER SYNDROME: 1.Oculomotor palsy (III radiations damage). 2.Contralateral Spastic Paralysis/Weakness (CST damage in cerebral peduncle). 3.Substantia Niagra damage (no symptoms due to no movement). **also called Superior Alternating Hemiparesis. |
What other two symptoms would you see with Weber Syndrome if they are not masked? | 1.Limb Ataxia (damage to cerebellarthalamic fibers). 2.Limb Dyskinesia (paladiothalamic fibers from Globus Palatus). |
Describe the presentation of a person with a vascular lesion in the Long Circumferential Territory | BENEDICT Syndrome: 1.Ipsilateral Occulomotor palsy (III Nuc damage). 2.Contralateral Sensory loss (ML damage). 3.Limb Ataxia (damage to cerebellarthalamic fibers). 4.Limb Dyskinesia (paladiothalamic fibers from Globus Palatus). |
How can you differentiate b/w Webers Syndrome and Benedict Syndrome since they BOTH present with Ipsilateral Occulomotor palsy, Limb dyskinesia, Limb ataxia? | BENEDICTS: sensory loss. WEBERS: motor loss. |
What is Parinaud Syndrome? How will the patient present? | Pineal Cyst compresses down onto the tectal region and the posterior commisure. This will disrupt the control of vertical gaze. **Patient will have Upgaze Palsy (inability to look up). |