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Anaphylaxis

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Question
Answer
Lymphocytes   show
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B cells   show
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show made out of proteins not cells  
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show maker molecules that identify them as foreign  
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Humoral immunity   show
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show respond only to specific organism  
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show killer T cells, Helper T cells, suppressor T cells  
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show does not involve antibodies, activate lymphocytes that attack and destroy foreign material  
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show once B cells and T cells have been activated by an antigen.  
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show ensure that the next time the body is exposed to the same antigen that produces the memory cell, the immune system kicks in to destroy it.  
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show immunity that naturally exist, it is not antigen specific, it does not need previous exposure to an antigen. Present from birth.  
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show Antigodies that comes from the mother to the baby. antibodies the newborn receives from the mother making the baby immune to the same microbes the mom is immune.  
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show from the mother to the baby in the breast milk to protect the baby's stomach  
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IgE   show
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show  
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show it is passed through mom to baby or it can be passes through serum obtained from other person who is immune to an specific infectious agent, protection for a few weeks.  
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active immunity   show
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show develops after exposure to specific antigens. , this immunity is acquired after B/T cells have been activated, slow response immunity, long lasting immunity, it can be active or passive.  
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show 1. non self recognition, responce to only foreign antigens.2. production of new antigen-specific antibodies. 3. memory cells produce antibody-produce lymphocyte for faster future response. 4. autoregulation for prevdention of tissue destruction,overreact  
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show injection, inhalation, absorption, ingestion  
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allergic reaction   show
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show exposure to antigens that can cause allergic reactions.  
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mast cells   show
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basophiles   show
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how do allergic reactions start   show
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show anahylactic reaction, mediated by IgE, caused by exogenous antigens, onset within mintues, initiated by antibidies, cells involved basophiles/eosinophiles.  
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type II hypersensitive reaction.   show
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type III hypersensitive reaction   show
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show cell mediated/delayes reactions, none antibody mediated, caused by tissue and organs antigens, onset 48-72hrs, lymphocytes and monocytes involved, initiated by T cells, appearance erythema/sclerosis  
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degranulation and substances released   show
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histamine   show
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show the most potent broncoconstrictor, causes wheezes, also coauses coronary vasoconstriction and increase vascular permeability  
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eosiniphil   show
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cardiovascular effects   show
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show N/V, diarrhea and severe abdominal pain and cramping may pccur in a patient having an anaphylactic reaction. increased GI activity.  
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show pt will have a feeling of impending doom due to the lack of gas exchange from the anaphylaxis, it may worse in confusion weakness, headache, syncope, seizures and coma  
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show the most visible sign that distinguish anahylaxis, signs caused by vasodilation induced by histamine. may see pruritus/erythema/urticaria/angioedema  
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pruritus   show
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erythema   show
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urticaria   show
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show significant swelling of the face, tounge, and deep tissues, reflect involvement of deep capillaries of the skin and mucus membranes  
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show autoimmune disease, caused by autoantibodies contribute to inflammation and damage of different organs and tissue thorughout the body. most common type of autoantibody is antinuclear antibodies (ANA)  
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lupus s/s   show
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lupus systemic effects   show
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show hardening of the skin, autoimmune that stimulates fibroblasts and increase the production of collagen. skin, organs and blood vessels can be affected.  
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show limited to the skin, and related tissues. localized scleroderma doesnt progress into systemic scleroderma.  
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raynauds phenomenon   show
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diffuse scleroderma   show
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show infection  
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show tissue from another person carrying non self markers and may be recognized as a foreign body  
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show donation of organs of people totally un related  
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show organ donation by identical twins  
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