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Inflammation

PATHO TEST 2

QuestionAnswer
Acute inflammation Rapid onset terminates quickly chemical mediators orchestrate response
Three stages of acute inflammation Increased vascular permeability - WBC platelets get through to inflammation (histamine) Cellular Chemotaxis - movement Systemic response
White Blood Response 1st 6-24 hours - Neutrophils - Replaced by monocytes 24-48 hours - monoctyes to macrophages - macro predominate in persistant inflammation - macro carry our phagocytosis Viral infections - lymphocytes dominate
Systemic responses in acute inflammation Fever lymphadenopathy anorexia sleepiness lethargy anemia weight loss chemical mediators - pyrogens, TNF-alpha, interleukins, drugs to reduce inflammation target these ^^
Fever Pyrogens cause fever WBCs and microorganisms release signals causing prostaglandins to reset temperature anti-prost reduce fever Fever onset - shivering to increase temp Fever "breaks" - sweating to reduce temperature
Reyes syndrome never give aspirin to children to reduce fever, cause swelling in brain
Outcomes of acute inflammation Complete resolution Healing by connective tissue -regeneration of normal cells does not occur -excessive proliferation of connective tissue, fibrous scar tissue formed Chronic inflammation -resolution does not occur -extensive tissue damage occur
Chronic Inflammation inflammation for weeks or months with no resolution or healing persistant infection - tuberculosis, syphilis, viruses hypersensitivity disorders - RA, SLE Exposure toxic agents - coal dust - anthracosis (black lung) Athersclerosis -chronic inflam. dis
Chronic differ from acute Predominance of monocytes, lymphocytes, and macrophages Continual secretion of cytokines damage healthy tissues stimulating further inflammation Granuloma formation - macrophages aggregate and are transformed into epithelial like cells
C vs A T and B lymphocytes amplify and perpetuate inflammatory signals - autoimmune disorders - EX: RA, Lupus
Created by: Mplaster
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